Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
about
A role for DEAD box 1 at DNA double-strand breaksRepair of endogenous DNA base lesions modulate lifespan in miceATM-dependent DNA damage-independent mitotic phosphorylation of H2AX in normally growing mammalian cells.PARP inhibition during alkylation-induced genotoxic stress signals a cell cycle checkpoint response mediated by ATM.The prion protein is critical for DNA repair and cell survival after genotoxic stress.Ataxia-telangiectasia: an inherited disorder of ionizing-radiation sensitivity in man. Progress in the elucidation of the underlying biochemical defect.Induction of base damages representing a high risk site for double-strand DNA break formation in genomic DNA by exposure of cells to DNA damaging agents.Efficient DNA base excision repair in ataxia telangiectasia cells.Modulation of proteostasis counteracts oxidative stress and affects DNA base excision repair capacity in ATM-deficient cells.
P2860
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P2860
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
description
1982 nî lūn-bûn
@nan
1982年の論文
@ja
1982年論文
@yue
1982年論文
@zh-hant
1982年論文
@zh-hk
1982年論文
@zh-mo
1982年論文
@zh-tw
1982年论文
@wuu
1982年论文
@zh
1982年论文
@zh-cn
name
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
@en
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
@nl
type
label
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
@en
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
@nl
prefLabel
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
@en
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
@nl
P4510
P1433
P1476
Hypersensitivity of ataxia telangiectasia skin fibroblasts to DNA alkylating agents.
@en
P2093
Barfknecht TR
P304
P356
10.1016/0027-5107(82)90299-8
P407
P577
1982-06-01T00:00:00Z