RHAMM is differentially expressed in the cell cycle and downregulated by the tumor suppressor p53.
about
Breaching the Castle Walls: Hyaluronan Depletion as a Therapeutic Approach to Cancer TherapyInteractions between Hyaluronan and Its Receptors (CD44, RHAMM) Regulate the Activities of Inflammation and CancerThe roles of hyaluronan/RHAMM/CD44 and their respective interactions along the insidious pathways of fibrosarcoma progressionThe p53-p21-DREAM-CDE/CHR pathway regulates G2/M cell cycle genesInterplay of mevalonate and Hippo pathways regulates RHAMM transcription via YAP to modulate breast cancer cell motilityInvestigating core genetic-and-epigenetic cell cycle networks for stemness and carcinogenic mechanisms, and cancer drug design using big database mining and genome-wide next-generation sequencing dataRHAMM deficiency disrupts folliculogenesis resulting in female hypofertility.The cytoskeletal protein RHAMM and ERK1/2 activity maintain the pluripotency of murine embryonic stem cells.Triptolide suppresses the in vitro and in vivo growth of lung cancer cells by targeting hyaluronan-CD44/RHAMM signaling.Prognostic impact of CD168 expression in gastric cancer.Proteomic analysis of potential keratan sulfate, chondroitin sulfate A, and hyaluronic acid molecular interactions.The transcriptional profile of mesenchymal stem cell populations in primary osteoporosis is distinct and shows overexpression of osteogenic inhibitorsp53 and cell cycle dependent transcription of kinesin family member 23 (KIF23) is controlled via a CHR promoter element bound by DREAM and MMB complexes.Assessing associations between the AURKA-HMMR-TPX2-TUBG1 functional module and breast cancer risk in BRCA1/2 mutation carriers.Receptor for hyaluronan-mediated motility isoform B promotes liver metastasis in a mouse model of multistep tumorigenesis and a tail vein assay for metastasis.Neural stem cell niches: roles for the hyaluronan-based extracellular matrix.Inhibition of hyaluronan synthase-3 decreases subcutaneous colon cancer growth by increasing apoptosisThe tumor suppressor p53 induces expression of the pregnancy-supporting human chorionic gonadotropin (hCG) CGB7 gene.Mechanism of MTA1 protein overexpression-linked invasion: MTA1 regulation of hyaluronan-mediated motility receptor (HMMR) expression and function.Emerging insights into the molecular and cellular basis of glioblastoma.RHAMM/HMMR (CD168) is not an ideal target antigen for immunotherapy of acute myeloid leukemia.The impact of post-transcriptional regulation in the p53 network.The Raf/MEK/ERK pathway can govern drug resistance, apoptosis and sensitivity to targeted therapy.Enhancing therapeutic efficacy by targeting non-oncogene addicted cells with combinations of signal transduction inhibitors and chemotherapyRHAMM splice variants confer radiosensitivity in human breast cancer cell linesHyaluronic acid promotes angiogenesis by inducing RHAMM-TGFβ receptor interaction via CD44-PKCδ.One function--multiple mechanisms: the manifold activities of p53 as a transcriptional repressor.Clinical peptide vaccination trials for leukemia patients.Glioblastoma, a brief review of history, molecular genetics, animal models and novel therapeutic strategies.Therapeutic targeting of hyaluronan in the tumor stroma.More than just a filler - the role of hyaluronan for skin homeostasis.Role of receptor for hyaluronan-mediated motility (RHAMM) in human head and neck cancers.Hyaluronan and RHAMM in wound repair and the "cancerization" of stromal tissues.Receptor for Hyaluronic Acid-Mediated Motility is Associated with Poor Survival in Pancreatic Ductal AdenocarcinomaHyaluronic acid in digestive cancers.Pomegranate juice and specific components inhibit cell and molecular processes critical for metastasis of breast cancer.The tumor-associated antigen RHAMM (HMMR/CD168) is expressed by monocyte-derived dendritic cells and presented to T cellsThe pancreatic cancer microenvironment: A true double agent.Increased RHAMM expression relates to ovarian cancer progression.Spindle Misorientation of Cerebral and Cerebellar Progenitors Is a Mechanistic Cause of Megalencephaly.
P2860
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P2860
RHAMM is differentially expressed in the cell cycle and downregulated by the tumor suppressor p53.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
RHAMM is differentially expres ...... d by the tumor suppressor p53.
@en
RHAMM is differentially expres ...... d by the tumor suppressor p53.
@nl
type
label
RHAMM is differentially expres ...... d by the tumor suppressor p53.
@en
RHAMM is differentially expres ...... d by the tumor suppressor p53.
@nl
prefLabel
RHAMM is differentially expres ...... d by the tumor suppressor p53.
@en
RHAMM is differentially expres ...... d by the tumor suppressor p53.
@nl
P356
P1433
P1476
RHAMM is differentially expres ...... d by the tumor suppressor p53.
@en
P2093
Sindy Sohr
P304
P356
10.4161/CC.7.21.7014
P577
2008-11-17T00:00:00Z