Induction of v-mos and activated Ha-ras oncogene expression in quiescent NIH 3T3 cells causes intracellular alkalinisation and cell-cycle progression.
about
Effect of cell spreading on cytoplasmic pH in normal and transformed fibroblastsDifferential occurrence of CSF-like activity and transforming activity of Mos during the cell cycle in fibroblastsGlycolysis, tumor metabolism, cancer growth and dissemination. A new pH-based etiopathogenic perspective and therapeutic approach to an old cancer questionHydrogen ion dynamics and the Na+/H+ exchanger in cancer angiogenesis and antiangiogenesis.Linking stem cells to chromosomal instability.Control of intracellular pH and growth by fibronectin in capillary endothelial cells.Antisense-fos RNA causes partial reversion of the transformed phenotypes induced by the c-Ha-ras oncogene.Na+/H+ exchanger NHE1 regulation modulates metastatic potential and epithelial-mesenchymal transition of triple-negative breast cancer cells.Cariporide and other new and powerful NHE1 inhibitors as potentially selective anticancer drugs--an integral molecular/biochemical/metabolic/clinical approach after one hundred years of cancer research.Role of pHi, and proton transporters in oncogene-driven neoplastic transformation.Acid-base transport in pancreatic cancer: molecular mechanisms and clinical potential.Regulation of intracellular pH in J774 murine macrophage cells: H+ extrusion processes.Naturally occurring deuterium is essential for the normal growth rate of cells.A Ca2(+)-activated K+ current in ras-transformed fibroblasts is absent from nontransformed cells.Transformation and pH homeostasis of fibroblasts expressing yeast H(+)-ATPase containing site-directed mutations.The role of calcium in cell shrinkage and intracellular alkalinization by bradykinin in Ha-ras oncogene expressing cells.Effects of bradykinin on cell volume and intracellular pH in NIH 3T3 fibroblasts expressing the ras oncogene.Interference of Ha-ras with inositol trisphosphate-mediated Ca(2+)-release.Na+/H+ antiporter gene expression increases during retinoic acid-induced granulocytic differentiation of HL60 cells.
P2860
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P2860
Induction of v-mos and activated Ha-ras oncogene expression in quiescent NIH 3T3 cells causes intracellular alkalinisation and cell-cycle progression.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Induction of v-mos and activat ...... on and cell-cycle progression.
@en
Induction of v-mos and activat ...... on and cell-cycle progression.
@nl
type
label
Induction of v-mos and activat ...... on and cell-cycle progression.
@en
Induction of v-mos and activat ...... on and cell-cycle progression.
@nl
prefLabel
Induction of v-mos and activat ...... on and cell-cycle progression.
@en
Induction of v-mos and activat ...... on and cell-cycle progression.
@nl
P2093
P1433
P1476
Induction of v-mos and activat ...... on and cell-cycle progression.
@en
P2093
P304
P356
10.1016/0378-1119(87)90357-X
P407
P577
1987-01-01T00:00:00Z