Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
about
Immunity to melanin and to tyrosinase in melanoma patients, and in people with vitiligo2-Ethoxybenzamide stimulates melanin synthesis in B16F1 melanoma cells via the CREB signaling pathway.Dual effects of acetylsalicylic acid on ERK signaling and Mitf transcription lead to inhibition of melanogenesis.The inhibitory effect of non-steroidal anti-inflammatory drugs (NSAIDs) on the monophenolase and diphenolase activities of mushroom tyrosinaseMolecular cloning and expression analysis of tyrosinase gene in the skin of Jining Gray Goat (Capra hircus).Indomethacin inhibits melanogenesis via down-regulation of Mitf mRNA transcription.
P2860
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年學術文章
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2010年學術文章
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name
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
@en
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
@nl
type
label
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
@en
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
@nl
prefLabel
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
@en
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
@nl
P2860
P1476
Depigmenting mechanism of NSAIDs on B16F1 melanoma cells.
@en
P2093
Hideki Takahashi
Masaru Toriyama
P2860
P2888
P304
P356
10.1007/S00403-010-1094-8
P50
P577
2010-11-16T00:00:00Z