Molecular mechanisms of regulation of fast-inactivating voltage-dependent transient outward K+ current in mouse heart by cell volume changes.
about
Heart-specific overexpression of the human short CLC-3 chloride channel isoform limits myocardial ischemia-induced ERP and QT prolongation.Resolution of hyposmotic stress in isolated mouse ventricular myocytes causes sealing of t-tubulesVolume regulation in the human airway epithelial cell line Calu-3.Double the keys, double the control: coupled phosphorylation sites provide novel molecular targets for precise control of ion channel function. Focus on "Differential regulation of a CLC anion channel by SPAK kinase ortholog-mediated multisite phosp
P2860
Molecular mechanisms of regulation of fast-inactivating voltage-dependent transient outward K+ current in mouse heart by cell volume changes.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Molecular mechanisms of regula ...... heart by cell volume changes.
@en
Molecular mechanisms of regula ...... heart by cell volume changes.
@nl
type
label
Molecular mechanisms of regula ...... heart by cell volume changes.
@en
Molecular mechanisms of regula ...... heart by cell volume changes.
@nl
prefLabel
Molecular mechanisms of regula ...... heart by cell volume changes.
@en
Molecular mechanisms of regula ...... heart by cell volume changes.
@nl
P2093
P2860
P1476
Molecular mechanisms of regula ...... heart by cell volume changes.
@en
P2093
Ge-Xin Wang
Guan-Lei Wang
Heather Baxter
Shintaro Yamamoto
P2860
P304
P356
10.1113/JPHYSIOL.2005.091264
P407
P577
2005-08-04T00:00:00Z