Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts. - Wikidata - awgldk - TriplyDB

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

http://www.wikidata.org/entity/Q42813301

about

Salicylate-induced degeneration of cochlea spiral ganglion neurons-apoptosis signaling An analysis of enzyme kinetics data for mitochondrial DNA strand termination by nucleoside reverse transcription inhibitors Decrease in hyperosmotic stress-induced corneal epithelial cell apoptosis by L-carnitine.Probing the nanoscale viscoelasticity of intracellular fluids in living cells.Rac1 mediates NaCl-induced superoxide generation in the thick ascending limb.Anoctamin 6 differs from VRAC and VSOAC but is involved in apoptosis and supports volume regulation in the presence of Ca2+.Possible causes of apoptotic volume decrease: an attempt at quantitative review.G protein independent phosphorylation and internalization of the delta-opioid receptor.Osmostress-induced apoptosis in Xenopus oocytes: role of stress protein kinases, calpains and Smac/DIABLO.An in vitro model of skeletal muscle volume regulation.Cell shrinkage and monovalent cation fluxes: role in apoptosis Mapping of hyperthermic tumor cell death in a microchannel under unidirectional heating.Ouabain-induced perturbations in intracellular ionic homeostasis regulate death receptor-mediated apoptosis.The apoptotic volume decrease is an upstream event of MAP kinase activation during Staurosporine-induced apoptosis in HeLa cells.Ethanol- and/or Taurine-Induced Oxidative Stress in Chick Embryos.A Systematic, Integrated Study on the Neuroprotective Effects of Hydroxysafflor Yellow A Revealed by (1)H NMR-Based Metabonomics and the NF-κB Pathway.NHE-1: a molecular target for signalling and cell matrix interactions.Sulforaphane decreases endothelial cell apoptosis in fuchs endothelial corneal dystrophy: a novel treatment.Synthesis, physicochemical characterization, and cytocompatibility of bioresorbable, dual-gelling injectable hydrogels.Apoptotic volume decrease as a geometric determinant for cell dismantling into apoptotic bodies.Cell volume homeostatic mechanisms: effectors and signalling pathways.Ion channels involved in cell volume regulation: effects on migration, proliferation, and programmed cell death in non adherent EAT cells and adherent ELA cells.Role of Rac1-mineralocorticoid-receptor signalling in renal and cardiac disease.Role of ion transport in control of apoptotic cell death.Ion channels and transporters in the development of drug resistance in cancer cells Physiological role of taurine--from organism to organelle.Analysis of the factors that limit the ability of feeder cells to maintain the undifferentiated state of human embryonic stem cells.Osmotic shrinkage elicits FAK- and Src phosphorylation and Src-dependent NKCC1 activation in NIH3T3 cells.Acquired cisplatin resistance in human ovarian A2780 cancer cells correlates with shift in taurine homeostasis and ability to volume regulate.Live fluorescence and transmission-through-dye microscopic study of actinomycin D-induced apoptosis and apoptotic volume decrease.Cofilin nuclear-cytoplasmic shuttling affects cofilin-actin rod formation during stress.ICB3E induces iNOS expression by ROS-dependent JNK and ERK activation for apoptosis of leukemic cells.Perturbation of intracellular K(+) homeostasis with valinomycin promotes cell death by mitochondrial swelling and autophagic processes.Potassium ion fluxes in corneal epithelial cells exposed to UVB.P2X4 activation modulates volume-sensitive outwardly rectifying chloride channels in rat hepatoma cells.Downregulation of LRRC8A protects human ovarian and alveolar carcinoma cells against Cisplatin-induced expression of p53, MDM2, p21Waf1/Cip1, and Caspase-9/-3 activation.Fate of hypertonicity-stressed corneal epithelial cells depends on differential MAPK activation and p38MAPK/Na-K-2Cl cotransporter1 interaction.Functional role of CCCTC binding factor (CTCF) in stress-induced apoptosis.Regulation of p53 in NIH3T3 mouse fibroblasts following hyperosmotic stress.Cell volume regulation and signaling in 3T3-L1 pre-adipocytes and adipocytes: on the possible roles of caveolae, insulin receptors, FAK and ERK1/2.

P2860

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P2860

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

http://www.wikidata.org/entity/Q42813301

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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2005年论文

@zh-cn

name

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

@en

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

@nl

type

label

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

@en

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

@nl

prefLabel

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

@en

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

@nl

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P356

JPHYSIOL.2005.087130

P1433

The Journal of Physiology

P1476

Cell shrinkage as a signal to apoptosis in NIH 3T3 fibroblasts.

@en

P2093

Christel R Friborg

http://www.w3.org/2001/XMLSchema#string

Else K Hoffmann

http://www.w3.org/2001/XMLSchema#string

Ian H Lambert

http://www.w3.org/2001/XMLSchema#string

Linda Schneider

http://www.w3.org/2001/XMLSchema#string

Maj-Britt Nielsen

http://www.w3.org/2001/XMLSchema#string

Martin B Friis

http://www.w3.org/2001/XMLSchema#string

P2860

Stress-induced inhibition of ERK1 and ERK2 by direct interaction with p38 MAP kinase

Activation of K+ channels induces apoptosis in vascular smooth muscle cells

p53 has a direct apoptogenic role at the mitochondria

Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis

Activation of transcription factor NF-kappaB and p38 mitogen-activated protein kinase is mediated by distinct and separate stress effector pathways

Signalling for survival and death in neurones: the role of stress-activated kinases, JNK and p38

Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

Potassium is a critical regulator of apoptotic enzymes in vitro and in vivo.

Role of p53 family members in apoptosis.

Normotonic cell shrinkage because of disordered volume regulation is an early prerequisite to apoptosis.

P304

427-443

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scholarly article

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10.1113/JPHYSIOL.2005.087130

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Pt 2

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567

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Søren T Christensen

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2005-06-23T00:00:00Z

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7767702

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15975986

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1474190

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