Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt.

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http://www.wikidata.org/entity/Q42820719

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2010 nî lūn-bûn

@nan

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

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2010年论文

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2010年论文

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name

Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt.

@en

Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt.

@nl

type

Item

label

Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt.

@en

Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt.

@nl

prefLabel

Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt.

@en

Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt.

@nl

P1476

Fas activation in adipocytes impairs insulin-stimulated glucose uptake by reducing Akt

@en

P2093

Eugen J Schoenle

http://www.w3.org/2001/XMLSchema#string

Reto A Rapold

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P2860

Deletion of Fas in adipocytes relieves adipose tissue inflammation and hepatic manifestations of obesity in mice.

Non-apoptotic Fas signaling.

Fas-beyond death: a regenerative role for Fas in the nervous system.

Fas ligand-induced proinflammatory transcriptional responses in reconstructed human epidermis. Recruitment of the epidermal growth factor receptor and activation of MAP kinases.

Basal lipolysis, not the degree of insulin resistance, differentiates large from small isolated adipocytes in high-fat fed mice.

An inhibitor of p38 mitogen-activated protein kinase prevents insulin-stimulated glucose transport but not glucose transporter translocation in 3T3-L1 adipocytes and L6 myotubes.

Interleukin-6 (IL-6) induces insulin resistance in 3T3-L1 adipocytes and is, like IL-8 and tumor necrosis factor-alpha, overexpressed in human fat cells from insulin-resistant subjects.

Tumor necrosis factor-{alpha} decreases Akt protein levels in 3T3-L1 adipocytes via the caspase-dependent ubiquitination of Akt.

Fas and Fas-associated death domain protein regulate monocyte chemoattractant protein-1 expression by human smooth muscle cells through caspase- and calpain-dependent release of interleukin-1alpha.

Isoform-specific regulation of insulin-dependent glucose uptake by Akt/protein kinase B.

P304

4187-4192

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P31

scholarly article

P356

10.1016/J.FEBSLET.2010.08.052

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

584

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P50

Daniel Konrad

Stephan Wueest

P577

2010-09-07T00:00:00Z

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P5875

46190662

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P698

20828573

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P921

Insulin