about
The host protein calprotectin modulates the Helicobacter pylori cag type IV secretion system via zinc sequestrationIdentification of an Acinetobacter baumannii zinc acquisition system that facilitates resistance to calprotectin-mediated zinc sequestrationTranslocator proteins in the two-partner secretion family have multiple domains.Nutritional immunity beyond iron: a role for manganese and zinc.Examination of type IV pilus expression and pilus-associated phenotypes in Kingella kingae clinical isolates.The CsoR-like sulfurtransferase repressor (CstR) is a persulfide sensor in Staphylococcus aureusControl of copper resistance and inorganic sulfur metabolism by paralogous regulators in Staphylococcus aureusExpression of Kingella kingae type IV pili is regulated by sigma54, PilS, and PilR.Role of copper efflux in pneumococcal pathogenesis and resistance to macrophage-mediated immune clearanceHost-imposed manganese starvation of invading pathogens: two routes to the same destination.Zinc sequestration by the neutrophil protein calprotectin enhances Salmonella growth in the inflamed gut.Inhibition of bacterial superoxide defense: a new front in the struggle between host and pathogen.A Superoxide Dismutase Capable of Functioning with Iron or Manganese Promotes the Resistance of Staphylococcus aureus to Calprotectin and Nutritional Immunity.Activation of heme biosynthesis by a small molecule that is toxic to fermenting Staphylococcus aureus.Cdc42 promotes host defenses against fatal infection.MntABC and MntH contribute to systemic Staphylococcus aureus infection by competing with calprotectin for nutrient manganeseLegionella pneumophila DotU and IcmF are required for stability of the Dot/Icm complex.Copper intoxication inhibits aerobic nucleotide synthesis in Streptococcus pneumoniae.Competition for Manganese at the Host-Pathogen Interface.Modulation of Kingella kingae adherence to human epithelial cells by type IV Pili, capsule, and a novel trimeric autotransporter.Nutrient metal sequestration by calprotectin inhibits bacterial superoxide defense, enhancing neutrophil killing of Staphylococcus aureus.Intracellular accumulation of staphylopine can sensitize Staphylococcus aureus to host-imposed zinc starvation by chelation-independent toxicityMetal-independent variants of phosphoglycerate mutase promote resistance to nutritional immunity and retention of glycolysis during infectionDisruption of Glycolysis by Nutritional Immunity Activates a Two-Component System That Coordinates a Metabolic and Antihost Response by Staphylococcus aureusAn evolutionary path to altered cofactor specificity in a metalloenzyme
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description
hulumtues
@sq
researcher
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wetenschapper
@nl
հետազոտող
@hy
name
Thomas E Kehl-Fie
@ast
Thomas E Kehl-Fie
@en
Thomas E Kehl-Fie
@es
Thomas E Kehl-Fie
@nl
Thomas E Kehl-Fie
@sl
type
label
Thomas E Kehl-Fie
@ast
Thomas E Kehl-Fie
@en
Thomas E Kehl-Fie
@es
Thomas E Kehl-Fie
@nl
Thomas E Kehl-Fie
@sl
prefLabel
Thomas E Kehl-Fie
@ast
Thomas E Kehl-Fie
@en
Thomas E Kehl-Fie
@es
Thomas E Kehl-Fie
@nl
Thomas E Kehl-Fie
@sl
P106
P21
P31
P496
0000-0001-8234-1113