5-HT2B receptors are expressed on astrocytes from brain and in culture and are a chronic target for all five conventional 'serotonin-specific reuptake inhibitors'.
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Signal Transduction in Astrocytes during Chronic or Acute Treatment with Drugs (SSRIs, Antibipolar Drugs, GABA-ergic Drugs, and Benzodiazepines) Ameliorating Mood DisordersFluoxetine affects GluK2 editing, glutamate-evoked Ca(2+) influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes.Chronic SSRI stimulation of astrocytic 5-HT2B receptors change multiple gene expressions/editings and metabolism of glutamate, glucose and glycogen: a potential paradigm shift.Fluoxetine and all other SSRIs are 5-HT2B Agonists - Importance for their Therapeutic Effects.Central role of glia in disease researchDisturbed development of the enteric nervous system after in utero exposure of selective serotonin re-uptake inhibitors and tricyclic antidepressants. Part 1: Literature review.Pronociceptive and Antinociceptive Effects of Buprenorphine in the Spinal Cord Dorsal Horn Cover a Dose Range of Four Orders of Magnitude.Decrease of gene expression of astrocytic 5-HT2B receptors parallels development of depressive phenotype in a mouse model of Parkinson's disease.Pharmacometabolomic mapping of early biochemical changes induced by sertraline and placebo.Antidepressants and Valvular Heart Disease: A Nested Case-Control Study in Taiwan.Cell type-specific gene expression and editing responses to chronic fluoxetine treatment in the in vivo mouse brain and their relevance for stress-induced anhedonia.5-HT2 ligands in the treatment of anxiety and depression.Astrocytic glycogenolysis: mechanisms and functions.Serotonin mediation of early memory formation via 5-HT2B receptor-induced glycogenolysis in the day-old chick.Sequential Astrocytic 5-HT2B Receptor Stimulation, [Ca(2+)]i Regulation, Glycogenolysis, Glutamate Synthesis, and K(+) Homeostasis are Similar but Not Identical in Learning and Mood Regulation.Response: Commentary: Chronic SSRI Stimulation of Astrocytic 5-HT2B Receptors Change Multiple Gene Expressions/Editings and Metabolism of Glutamate, Glucose and Glycogen: A Potential Paradigm Shift.Commentary: Chronic SSRI stimulation of astrocytic 5-HT2B receptors change multiple gene expressions/editings and metabolism of glutamate, glucose and glycogen: a potential paradigm shiftBiphasic Regulation of Caveolin-1 Gene Expression by Fluoxetine in Astrocytes: Opposite Effects of PI3K/AKT and MAPK/ERK Signaling Pathways on c-fos.Physiology of Astroglia.
P2860
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P2860
5-HT2B receptors are expressed on astrocytes from brain and in culture and are a chronic target for all five conventional 'serotonin-specific reuptake inhibitors'.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
5-HT2B receptors are expressed ...... specific reuptake inhibitors'.
@en
5-HT2B receptors are expressed ...... specific reuptake inhibitors'.
@nl
type
label
5-HT2B receptors are expressed ...... specific reuptake inhibitors'.
@en
5-HT2B receptors are expressed ...... specific reuptake inhibitors'.
@nl
prefLabel
5-HT2B receptors are expressed ...... specific reuptake inhibitors'.
@en
5-HT2B receptors are expressed ...... specific reuptake inhibitors'.
@nl
P2093
P2860
P50
P1433
P1476
5-HT2B receptors are expressed ...... specific reuptake inhibitors'.
@en
P2093
Ditte Lovatt
Liang Peng
Shiquen Zhang
Steven A Goldman
P2860
P304
P356
10.1017/S1740925X10000141
P577
2010-05-01T00:00:00Z