Acetylcholine acutely modifies nitric oxide synthase function in the human coronary circulation.

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The potential role of endothelial dysfunction and platelet activation in the development of thrombotic risk in COPD patients.

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Acetylcholine acutely modifies nitric oxide synthase function in the human coronary circulation.

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2010 nî lūn-bûn

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Acetylcholine acutely modifies ...... he human coronary circulation.

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Acetylcholine acutely modifies ...... he human coronary circulation.

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Acetylcholine acutely modifies ...... he human coronary circulation.

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Acetylcholine acutely modifies ...... the human coronary circulation

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Abdul Al-Hesayen

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Lynne E Nield

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Steven E S Miner

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Tommaso Gori

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Nitric oxide activity is deficient in spasm arteries of patients with coronary spastic angina

Relation of Basal coronary nitric oxide activity to the burden of atherosclerosis.

Endothelial regulation of vasomotion in apoE-deficient mice: implications for interactions between peroxynitrite and tetrahydrobiopterin.

Abnormal coronary vasomotion as a prognostic indicator of cardiovascular events in women: results from the National Heart, Lung, and Blood Institute-Sponsored Women's Ischemia Syndrome Evaluation (WISE).

Is the fatty meal a trigger for acute coronary syndromes.

Atherosclerosis influences the vasomotor response of epicardial coronary arteries to exercise.

Endothelium-derived hyperpolarizing factor but not NO reduces smooth muscle Ca2+ during acetylcholine-induced dilation of microvessels.

Role of oxidative stress in atherosclerosis.

Restoration of endothelium-dependent vasodilation after reperfusion injury by tetrahydrobiopterin.

Inhibition of constitutive nitric oxide synthase (NOS) by nitric oxide generated by inducible NOS after lipopolysaccharide administration provokes renal dysfunction in rats.

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10.1113/EXPPHYSIOL.2010.053926

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John D. Parker

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2010-09-03T00:00:00Z

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Acetylcholine acutely modifies nitric oxide synthase function in the human coronary circulation.

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Acetylcholine acutely modifies nitric oxide synthase function in the human coronary circulation.

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