PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

Item

http://www.wikidata.org/entity/Q42920788

description

2009 nî lūn-bûn

@nan

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

2009年论文

@zh

2009年论文

@zh-cn

name

PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

@en

PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

@nl

type

Item

label

PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

@en

PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

@nl

prefLabel

PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

@en

PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

@nl

P1476

PACAP/PAC1R signaling modulates acetylcholine release at neuronal nicotinic synapses.

@en

P2093

Joseph F Margiotta

http://www.w3.org/2001/XMLSchema#string

Selwyn S Jayakar

http://www.w3.org/2001/XMLSchema#string

P2860

The dual effect of membrane potential on sodium conductance in the giant axon of Loligo

Isolation of nitric oxide synthetase, a calmodulin-requiring enzyme

Quantal components of the end-plate potential

Nitric oxide synthase regulatory sites. Phosphorylation by cyclic AMP-dependent protein kinase, protein kinase C, and calcium/calmodulin protein kinase; identification of flavin and calmodulin binding sites

Assembly of the N-cadherin complex during synapse formation involves uncoupling of p120-catenin and association with presenilin 1

RMI 12330 A, an inhibitor of adenylate cyclase in rat liver

Kv4 potassium channel subunits control action potential repolarization and frequency-dependent broadening in rat hippocampal CA1 pyramidal neurones

Pituitary adenylyl cyclase-activating polypeptide (PACAP) and its receptor (PAC1-R) are positioned to modulate afferent signaling in the cochlea

A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde m

Developmental changes in the structure of the synapse on the myelinated cell bodies of the chicken ciliary ganglion

P304

244-257

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.MCN.2009.11.007

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Phyllis Pugh

P577

2009-12-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

40445854

http://www.w3.org/2001/XMLSchema#string

P698

19958833

http://www.w3.org/2001/XMLSchema#string

P932

2818583

http://www.w3.org/2001/XMLSchema#string