Further evidence of IBD5/CARD15 (NOD2) epistasis in the susceptibility to ulcerative colitis. - Wikidata - awgldk - TriplyDB

Further evidence of IBD5/CARD15 (NOD2) epistasis in the susceptibility to ulcerative colitis.

Further evidence of IBD5/CARD15 (NOD2) epistasis in the susceptibility to ulcerative colitis.

http://www.wikidata.org/entity/Q42927789

about

Role of discs large homolog 5 OCTNs: will the real IBD5 gene please stand up?Single nucleotide polymorphisms of OCTN1, OCTN2, and DLG5 genes in Greek patients with Crohn's disease Role of CARD15, DLG5 and OCTN genes polymorphisms in children with inflammatory bowel diseases.Novel genetic markers in inflammatory bowel disease.Polymorphisms in the DLG5 and OCTN cation transporter genes in Crohn's disease.Evidence for association of OCTN genes and IBD5 with ulcerative colitis Analysis of the influence of OCTN1/2 variants within the IBD5 locus on disease susceptibility and growth indices in early onset inflammatory bowel disease NOD2/CARD15 gene mutations in North Algerian patients with inflammatory bowel disease.Clinical implications of inflammatory bowel disease genetics on phenotype.Genetics of Crohn disease, an archetypal inflammatory barrier disease.Current status of genetics research in inflammatory bowel disease.Role of the IBD5 susceptibility locus in the inflammatory bowel diseases.Role of genes, the environment and their interactions in the etiology of inflammatory bowel diseases.Genetics of inflammatory bowel disease: current status and future directions.A critical analysis of barriers to the clinical implementation of pharmacogenomics.Genetics of the innate immune response in inflammatory bowel disease.NOD2/CARD15 disease associations other than Crohn's disease.NOD-like receptors and inflammation.Susceptibility to ulcerative colitis in Hungarian patients determined by gene-gene interactions.New insights into gastrointestinal and hepatic granulomatous disorders.The association between autoimmunity and pouchitis.A population-based case-control study of CARD15 and other risk factors in Crohn's disease and ulcerative colitis.Phenotype-stratified genetic linkage study demonstrates that IBD2 is an extensive ulcerative colitis locus.Lack of association between IBD5 and Crohn's disease in Japanese patients demonstrates population-specific differences in inflammatory bowel disease.Refined genomic localization and ethnic differences observed for the IBD5 association with Crohn's disease Contribution of IBD5 Locus to Clinical Features of IBD Patients

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P2860

Further evidence of IBD5/CARD15 (NOD2) epistasis in the susceptibility to ulcerative colitis.

http://www.wikidata.org/entity/Q42927789

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

@zh-hant

2003年論文

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2003年論文

@zh-mo

2003年論文

@zh-tw

2003年论文

@wuu

2003年论文

@zh

2003年论文

@zh-cn

name

Further evidence of IBD5/CARD1 ...... ibility to ulcerative colitis.

@en

Further evidence of IBD5/CARD15

@nl

type

label

Further evidence of IBD5/CARD1 ...... ibility to ulcerative colitis.

@en

Further evidence of IBD5/CARD15

@nl

prefLabel

Further evidence of IBD5/CARD1 ...... ibility to ulcerative colitis.

@en

Further evidence of IBD5/CARD15

@nl

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American Journal of Human Genetics

P1476

Further evidence of IBD5/CARD1 ...... ibility to ulcerative colitis.

@en

P2093

Derek P Jewell

http://www.w3.org/2001/XMLSchema#string

Dermot P B McGovern

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Kenichi Negoro

http://www.w3.org/2001/XMLSchema#string

Tariq Ahmad

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P2860

A novel NOD2/CARD15 haplotype conferring risk for Crohn disease in Ashkenazi Jews

Genetic evidence for interaction of the 5q31 cytokine locus and the CARD15 gene in Crohn disease.

IBD5 is a general risk factor for inflammatory bowel disease: replication of association with Crohn disease and identification of a novel association with ulcerative colitis.

CARD15: a pleiotropic autoimmune gene that confers susceptibility to psoriatic arthritis.

Analysis of the IBD5 locus and potential gene-gene interactions in Crohn's disease.

Genotype-phenotype analysis of the Crohn's disease susceptibility haplotype on chromosome 5q31.

Crohn's disease-associated NOD2 variants share a signaling defect in response to lipopolysaccharide and peptidoglycan.

P304

1465-1466

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scholarly article

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10.1086/379745

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6

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73

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David A van Heel

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2003-12-01T00:00:00Z

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8976874

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1180410

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