Regression mapping of association between the human leukocyte antigen region and Graves disease. - Wikidata - awgldk - TriplyDB

Regression mapping of association between the human leukocyte antigen region and Graves disease.

Regression mapping of association between the human leukocyte antigen region and Graves disease.

http://www.wikidata.org/entity/Q42949415

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The interferon induced with helicase domain 1 A946T polymorphism is not associated with rheumatoid arthritis Follow-up of potential novel Graves' disease susceptibility loci, identified in the UK WTCCC genome-wide nonsynonymous SNP study The CD40, CTLA-4, thyroglobulin, TSH receptor, and PTPN22 gene quintet and its contribution to thyroid autoimmunity: back to the future Interrogating the major histocompatibility complex with high-throughput genomics Mechanisms of autoimmune thyroid diseases: from genetics to epigenetics HLA-DRB1*07:01 is associated with a higher risk of asparaginase allergies.Identification of two independent risk factors for lupus within the MHC in United Kingdom families.Evidence for association between the HLA-DQA locus and abdominal aortic aneurysms in the Belgian population: a case control study Association of 4p14 and 6q27 variation with Graves disease: a case-control study and a meta-analysis of available evidence.Association between IL-21 gene rs907715 polymorphisms and Graves' disease in a Southern Chinese population.Polymorphisms of CLEC16A region and autoimmune thyroid diseases.Comprehensive genotyping in two homogeneous Graves' disease samples reveals major and novel HLA association alleles.Shared genetic relationships underlying generalized vitiligo and autoimmune thyroid disease.Genetic susceptibility to autoimmune thyroid disease: past, present, and future Genetics of thyroid function and disease.Regulation of major histocompatibility complex class II gene expression, genetic variation and disease.The association between HLA class II haplotype with Graves' disease in Thai population.Use of Tag single nucleotide polymorphisms (SNPs) to screen PTPN21: no association with Graves' disease.A refined study of FCRL genes from a genome-wide association study for Graves' disease.Genetic heterogeneity of susceptibility gene in different ethnic populations: refining association study of PTPN22 for Graves' disease in a Chinese Han population.Genetic factors of autoimmune thyroid diseases in Japanese Immunogenetic mechanisms leading to thyroid autoimmunity: recent advances in identifying susceptibility genes and regions.The genetic basis of graves' disease.Role of major histocompatibility complex class II in the development of autoimmune type 1 diabetes and thyroiditis in rats Identifying a Small Molecule Blocking Antigen Presentation in Autoimmune Thyroiditis.Shared Genetic Basis for Type 1 Diabetes, Islet Autoantibodies, and Autoantibodies Associated With Other Immune-Mediated Diseases in Families With Type 1 Diabetes Immunogenetics of autoimmune thyroid diseases: A comprehensive review.The etiology of autoimmune thyroid disease: a story of genes and environment A type 1 diabetes subgroup with a female bias is characterised by failure in tolerance to thyroid peroxidase at an early age and a strong association with the cytotoxic T-lymphocyte-associated antigen-4 gene Genetic and epigenetic mechanisms in thyroid autoimmunity.Early-onset, coexisting autoimmunity and decreased HLA-mediated susceptibility are the characteristics of diabetes in Down syndrome.Joint genetic susceptibility to type 1 diabetes and autoimmune thyroiditis: from epidemiology to mechanisms.Recent insights into the pathogenesis and management of thyroid-associated ophthalmopathy Genetic developments in autoimmune thyroid disease: an evolutionary process.The HLA gene complex in thyroid autoimmunity: from epidemiology to etiology.HLA DRB1*03 as a possible common etiology of schizophrenia, Graves' disease, and type 2 diabetes.Shared genetics in coeliac disease and other immune-mediated diseases.The search for the genetic contribution to autoimmune thyroid disease: the never ending story?The role of glutamic or aspartic acid in position four of the epitope binding motif and thyrotropin receptor-extracellular domain epitope selection in Graves' disease.Identification of BACH2 as a susceptibility gene for Graves' disease in the Chinese Han population based on a three-stage genome-wide association study.

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P2860

Regression mapping of association between the human leukocyte antigen region and Graves disease.

http://www.wikidata.org/entity/Q42949415

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Regression mapping of associat ...... gen region and Graves disease.

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Regression mapping of associat ...... gen region and Graves disease.

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type

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Regression mapping of associat ...... gen region and Graves disease.

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Regression mapping of associat ...... gen region and Graves disease.

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Regression mapping of associat ...... gen region and Graves disease.

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Regression mapping of associat ...... gen region and Graves disease.

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American Journal of Human Genetics

P1476

Regression mapping of associat ...... gen region and Graves disease.

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Heather J Cordell

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Helen Foxall

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Jackie Carr-Smith

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Jayne A Franklyn

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Joanna M M Howson

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Joanne M Heward

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Matthew J Simmonds

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Sarah M Gibson

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Stephen C L Gough

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Yaron Tomer

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P2860

Association of the T-cell regulatory gene CTLA4 with susceptibility to autoimmune disease

The shared epitope hypothesis. An approach to understanding the molecular genetics of susceptibility to rheumatoid arthritis

A correlation between the relative predisposition of MHC class II alleles to type 1 diabetes and the structure of their proteins.

A unified stepwise regression procedure for evaluating the relative effects of polymorphisms within a gene using case/control or family data: application to HLA in type 1 diabetes

HLA-DRB1*08, DRB1*03/DRB3*0101, and DRB3*0202 are susceptibility genes for Graves' disease in North American Caucasians, whereas DRB1*07 is protective.

The genetics of Graves' disease.

Correlation between disease phenotype and genetic heterogeneity in rheumatoid arthritis.

HLA antigens in Graves' disease.

Autoimmune thyroid disease: further developments in our understanding.

Point mutations in or near the antigen-binding groove of HLA-DR3 implicate class II-associated invariant chain peptide affinity as a constraint on MHC class II polymorphism.

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157-163

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10.1086/426947

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1

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2004-11-22T00:00:00Z

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8167878

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15558498

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1196419

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