Creatine kinase-overexpression improves myocardial energetics, contractile dysfunction and survival in murine doxorubicin cardiotoxicity.
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Recent Advances on Pathophysiology, Diagnostic and Therapeutic Insights in Cardiac Dysfunction Induced by Antineoplastic DrugsMyocardial creatine levels do not influence response to acute oxidative stress in isolated perfused heartModular organization of cardiac energy metabolism: energy conversion, transfer and feedback regulation.MRS: a noninvasive window into cardiac metabolism.Increasing creatine kinase activity protects against hypoxia / reoxygenation injury but not against anthracycline toxicity in vitro.Metabolic remodelling in hypertrophied and failing myocardium: a review.Creatine supplementation reduces doxorubicin-induced cardiomyocellular injury.Enzyme-Responsive LipoCEST Agents: Assessment of MMP-2 Activity by Measuring the Intra-liposomal Water 1 H NMR Shift.Antineoplastic Drug-Induced Cardiotoxicity: A Redox Perspective.
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P2860
Creatine kinase-overexpression improves myocardial energetics, contractile dysfunction and survival in murine doxorubicin cardiotoxicity.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
Creatine kinase-overexpression ...... ne doxorubicin cardiotoxicity.
@en
Creatine kinase-overexpression ...... ne doxorubicin cardiotoxicity.
@nl
type
label
Creatine kinase-overexpression ...... ne doxorubicin cardiotoxicity.
@en
Creatine kinase-overexpression ...... ne doxorubicin cardiotoxicity.
@nl
prefLabel
Creatine kinase-overexpression ...... ne doxorubicin cardiotoxicity.
@en
Creatine kinase-overexpression ...... ne doxorubicin cardiotoxicity.
@nl
P2093
P2860
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P1476
Creatine kinase-overexpression ...... ne doxorubicin cardiotoxicity.
@en
P2093
Ashish Gupta
Charles Steenbergen
Cory Rohlfsen
Michelle K Leppo
Robert G Weiss
Vadappuram P Chacko
P2860
P304
P356
10.1371/JOURNAL.PONE.0074675
P407
P50
P577
2013-10-01T00:00:00Z