Hepatic ISG expression is associated with genetic variation in interleukin 28B and the outcome of IFN therapy for chronic hepatitis C. - Wikidata - awgldk - TriplyDB

Hepatic ISG expression is associated with genetic variation in interleukin 28B and the outcome of IFN therapy for chronic hepatitis C.

Hepatic ISG expression is associated with genetic variation in interleukin 28B and the outcome of IFN therapy for chronic hepatitis C.

http://www.wikidata.org/entity/Q42996760

about

IFNL3 mRNA structure is remodeled by a functional non-coding polymorphism associated with hepatitis C virus clearance A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus Hepatitis C virus protease inhibitor-resistance mutations: our experience and review Interferon Response in Hepatitis C Virus (HCV) Infection: Lessons from Cell Culture Systems of HCV Infection Impact of IL28B, ITPA and PNPLA3 genetic variants on therapeutic outcome and progression of hepatitis C virus infection Regulation of hepatic innate immunity by hepatitis C virus Immune responses to HCV and other hepatitis viruses Emerging concepts in immunity to hepatitis C virus infection.Genetic variants at the IFNL3 locus and their association with hepatitis C virus infections reveal novel insights into host-virus interactions Innate immune responses in hepatitis C virus infection The Role of Interferon Lambda 3 Genetic Polymorphisms in Response to Interferon Therapy in Chronic Hepatitis B Patients: An Updated Meta-Analysis Interferon-λ4 is a cell-autonomous type III interferon associated with pre-treatment hepatitis C virus burden IL28B, HLA-C, and KIR variants additively predict response to therapy in chronic hepatitis C virus infection in a European Cohort: a cross-sectional study Dysregulation of IFN system can lead to poor response to pegylated interferon and ribavirin therapy in chronic hepatitis C Identification of improved IL28B SNPs and haplotypes for prediction of drug response in treatment of hepatitis C using massively parallel sequencing in a cross-sectional European cohort Relation of IL28B gene polymorphism with biochemical and histological features in hepatitis C virus-induced liver disease Association of IFNL3 rs12979860 and rs8099917 with biochemical predictors of interferon responsiveness in chronic hepatitis C virus infection Impact of IL28B-related single nucleotide polymorphisms on liver histopathology in chronic hepatitis C genotype 2 and 3 Hepatic interferon-stimulated genes are differentially regulated in the liver of chronic hepatitis C patients with different interleukin-28B genotypes.Pharmacogenetics of hepatitis C: transition from interferon-based therapies to direct-acting antiviral agents Ex vivo induction of IFN-λ3 by a TLR7 agonist determines response to Peg-IFN/ribavirin therapy in chronic hepatitis C patients.microRNA-122 abundance in hepatocellular carcinoma and non-tumor liver tissue from Japanese patients with persistent HCV versus HBV infection.Impact of donor and recipient single nucleotide polymorphisms of IL28B rs8099917 in living donor liver transplantation for hepatitis C Genome-wide association study of interferon-related cytopenia in chronic hepatitis C patients.Effect of laparoscopic splenectomy in patients with Hepatitis C and cirrhosis carrying IL28B minor genotype IFN-λ receptor 1 expression is induced in chronic hepatitis C and correlates with the IFN-λ3 genotype and with nonresponsiveness to IFN-α therapies IFNL3/4 genotype is associated with altered immune cell populations in peripheral blood in chronic hepatitis C infection Involvement of MAP3K8 and miR-17-5p in poor virologic response to interferon-based combination therapy for chronic hepatitis C.Protective immunity against hepatitis C: many shades of gray.Response prediction in chronic hepatitis C by assessment of IP-10 and IL28B-related single nucleotide polymorphisms.No association between the IL28B SNP and response to peginterferon plus ribavirin combination treatment in Korean chronic hepatitis C patients.Predicting response to therapy in chronic hepatitis C: an approach combining interleukin-28B gene polymorphisms and clinical data.Characterization of serum proteins associated with IL28B genotype among patients with chronic hepatitis C.USP18-based negative feedback control is induced by type I and type III interferons and specifically inactivates interferon α response.Hepatitis C virus in the new era: perspectives in epidemiology, prevention, diagnostics and predictors of response to therapy.Hepatitis C virus infection suppresses the interferon response in the liver of the human hepatocyte chimeric mouse An interferon response gene signature is associated with the therapeutic response of hepatitis C patients IL28B alleles exert an additive dose effect when applied to HCV-HIV coinfected persons undergoing peginterferon and ribavirin therapy Single nucleotide polymorphisms of the IL28B and sustained virologic response of patients with chronic hepatitis C to PEG-interferon/ribavirin therapy: A meta-analysis: Meta-analysis of IL28B.Hepatic STAT1-nuclear translocation and interleukin 28B polymorphisms predict treatment outcomes in hepatitis C virus genotype 1-infected patients.

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P2860

Hepatic ISG expression is associated with genetic variation in interleukin 28B and the outcome of IFN therapy for chronic hepatitis C.

http://www.wikidata.org/entity/Q42996760

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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2010年學術文章

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name

Hepatic ISG expression is asso ...... erapy for chronic hepatitis C.

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Hepatic ISG expression is asso ...... erapy for chronic hepatitis C.

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type

label

Hepatic ISG expression is asso ...... erapy for chronic hepatitis C.

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Hepatic ISG expression is asso ...... erapy for chronic hepatitis C.

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prefLabel

Hepatic ISG expression is asso ...... erapy for chronic hepatitis C.

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Hepatic ISG expression is asso ...... erapy for chronic hepatitis C.

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J.GASTRO.2010.04.049

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Hepatic ISG expression is asso ...... erapy for chronic hepatitis C.

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Akito Sakai

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Eishiro Mizukoshi

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Hokuriku Liver Study Group

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Katsuhisa Horimoto

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Katsushi Tokunaga

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Masao Honda

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Masashi Mizokami

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Mikiko Nakamura

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Shuichi Kaneko

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Takayoshi Shirasaki

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499-509

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scholarly article

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10.1053/J.GASTRO.2010.04.049

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