p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation. - Wikidata - awgldk - TriplyDB

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

http://www.wikidata.org/entity/Q43003484

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p38 mitogen-activated protein kinase controls NF-kappaB transcriptional activation and tumor necrosis factor alpha production through RelA phosphorylation mediated by mitogen- and stress-activated protein kinase 1 in response to Borrelia burgdorferi Differential effects of ERK and p38 signaling in BMP-2 stimulated hypertrophy of cultured chick sternal chondrocytes BMP canonical Smad signaling through Smad1 and Smad5 is required for endochondral bone formation Cytokine-like 1 (Cytl1) regulates the chondrogenesis of mesenchymal cells Mice with an N-Ethyl-N-Nitrosourea (ENU) Induced Tyr209Asn Mutation in Natriuretic Peptide Receptor 3 (NPR3) Provide a Model for Kyphosis Associated with Activation of the MAPK Signaling Pathway p38 MAPK mediated in compressive stress-induced chondrogenesis of rat bone marrow MSCs in 3D alginate scaffolds Inactivation of Patched1 in the mouse limb has novel inhibitory effects on the chondrogenic program.The transcription factor ATF3 is upregulated during chondrocyte differentiation and represses cyclin D1 and A gene transcription Dexamethasone stimulates expression of C-type Natriuretic Peptide in chondrocytes.Expression profiling of Dexamethasone-treated primary chondrocytes identifies targets of glucocorticoid signalling in endochondral bone development.The PI3K pathway regulates endochondral bone growth through control of hypertrophic chondrocyte differentiation.Regulation of gene expression by PI3K in mouse growth plate chondrocytes.GADD45beta enhances Col10a1 transcription via the MTK1/MKK3/6/p38 axis and activation of C/EBPbeta-TAD4 in terminally differentiating chondrocytes High-content drug screening with engineered musculoskeletal tissues p38 MAPK regulates IL-1beta induced IL-6 expression through mRNA stability in osteoblasts Aberrant hypertrophy in Smad3-deficient murine chondrocytes is rescued by restoring transforming growth factor beta-activated kinase 1/activating transcription factor 2 signaling: a potential clinical implication for osteoarthritis.Microarray analyses of gene expression during chondrocyte differentiation identifies novel regulators of hypertrophy.Constitutive activation of MKK6 in chondrocytes of transgenic mice inhibits proliferation and delays endochondral bone formation Reactive oxygen species generated by NADPH oxidase 2 and 4 are required for chondrogenic differentiation.Novel arylpyrazole compounds selectively modulate glucocorticoid receptor regulatory activity.Mice with a conditional deletion of the neurotrophin receptor TrkB are dwarfed, and are similar to mice with a MAPK14 deletion Mitogen-activated protein kinase p38 induces HDAC4 degradation in hypertrophic chondrocytes.Skeletal cell differentiation is enhanced by atmospheric dielectric barrier discharge plasma treatment Strategies to minimize hypertrophy in cartilage engineering and regeneration.Biomimetic 3D Tissue Models for Advanced High-Throughput Drug Screening.Phosphate stimulates matrix Gla protein expression in chondrocytes through the extracellular signal regulated kinase signaling pathway Coculture strategies in bone tissue engineering: the impact of culture conditions on pluripotent stem cell populations.BDNF alters ERK/p38 MAPK activity ratios to promote differentiation in growth plate chondrocytes Spatial and temporal regulation of gene expression in the mammalian growth plate.Src kinase inhibition promotes the chondrocyte phenotype.p38α MAPK Regulates Lineage Commitment and OPG Synthesis of Bone Marrow Stromal Cells to Prevent Bone Loss under Physiological and Pathological Conditions.Murine and chicken chondrocytes regulate osteoclastogenesis by producing RANKL in response to BMP2 Sonic hedgehog signaling directly targets Hyaluronic Acid Synthase 2, an essential regulator of phalangeal joint patterning.GSK-3β function in bone regulates skeletal development, whole-body metabolism, and male life span PPARgamma2 expression in growth plate chondrocytes is regulated by p38 and GSK-3.MicroRNA-140 Provides Robustness to the Regulation of Hypertrophic Chondrocyte Differentiation by the PTHrP-HDAC4 Pathway.The myogenic kinome: protein kinases critical to mammalian skeletal myogenesis.Focus on the p38 MAPK signaling pathway in bone development and maintenance.Focal adhesion kinase/Src suppresses early chondrogenesis: central role of CCN2.Vinculin functions as regulator of chondrogenesis

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P2860

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

http://www.wikidata.org/entity/Q43003484

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

@en

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

@nl

type

label

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

@en

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

@nl

prefLabel

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

@en

p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

@nl

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Biochemical Journal

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p38 MAP kinase signalling is required for hypertrophic chondrocyte differentiation.

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P2093

Arthur V Sampaio

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Lee-Anne Stanton

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Shalev Sabari

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T Michael Underhill

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P2860

Regulation of the MEF2 family of transcription factors by p38

Targeting of p38 mitogen-activated protein kinases to MEF2 transcription factors

Fibroblast growth factor receptor 3 is a negative regulator of bone growth

GATA-dependent recruitment of MEF2 proteins to target promoters

Articular cartilage and changes in arthritis. An introduction: cell biology of osteoarthritis

Toward understanding SOX9 function in chondrocyte differentiation

Chondrodysplasia and neurological abnormalities in ATF-2-deficient mice

Pro-inflammatory cytokines and environmental stress cause p38 mitogen-activated protein kinase activation by dual phosphorylation on tyrosine and threonine

In situ hybridization studies on the expression of type X collagen in fetal human cartilage

Requirement for p38alpha in erythropoietin expression: a role for stress kinases in erythropoiesis

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Pt 1

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378

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