Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells. - Wikidata - awgldk - TriplyDB

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

http://www.wikidata.org/entity/Q43050126

about

Evidence for a telomere-independent "clock" limiting RAS oncogene-driven proliferation of human thyroid epithelial cells Acrylamide: review of toxicity data and dose-response analyses for cancer and noncancer effects.Recent developments in the investigation of thyroid regulation and thyroid carcinogenesis In vitro responsiveness to serum growth factors is inversely related to in vivo malignancy in human thyroid epithelial cells.The insulin and igf-I pathway in endocrine glands carcinogenesis Abnormalities of the EGF receptor system in human thyroid neoplasia.Control of replicative life span in human cells: barriers to clonal expansion intermediate between M1 senescence and M2 crisis.Mutant ras-induced proliferation of human thyroid epithelial cells requires three effector pathways.RAS oncogene activation induces proliferation in normal human thyroid epithelial cells without loss of differentiation.Activation of mitogen-activated protein kinase is necessary but not sufficient for proliferation of human thyroid epithelial cells induced by mutant Ras.Contrasting effects of activating mutations of GalphaS and the thyrotropin receptor on proliferation and differentiation of thyroid follicular cells.Spontaneous de-differentiation correlates with extended lifespan in transformed thyroid epithelial cells: an epigenetic mechanism of tumour progression?Expression of c-erbB-2 protein in papillary thyroid carcinomas.Does autocrine growth factor secretion form part of a mechanism which paradoxically protects against tumour development?Evidence for mutual interdependence of epithelium and stromal lymphoid cells in a subset of papillary carcinomas.Cytofluorometric analysis of lymphocyte subsets in thyroid aspirates from patients with autonomously functioning nodules.Opposing effects of mutant ras oncoprotein on human fibroblast and epithelial cell proliferation: implications for models of human tumorigenesis.Co-expression of the genes encoding transforming growth factor-alpha and its receptor in papillary carcinomas of the thyroid.Molecular mechanisms of human thyrocyte dysfunction induced by low concentrations of polychlorinated biphenyl 118 through the Akt/FoxO3a/NIS pathway.Insulin-Like Growth Factor I in Human Thyroid Tissue: Specific Localization by Immunohistochemistry and In Situ Hybridization.

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P2860

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

http://www.wikidata.org/entity/Q43050126

description

1988 nî lūn-bûn

@nan

1988年の論文

@ja

1988年学术文章

@wuu

1988年学术文章

@zh

1988年学术文章

@zh-cn

1988年学术文章

@zh-hans

1988年学术文章

@zh-my

1988年学术文章

@zh-sg

1988年學術文章

@yue

1988年學術文章

@zh-hant

name

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

@en

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

@nl

type

label

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

@en

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

@nl

prefLabel

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

@en

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

@nl

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British Journal of Cancer

P1476

Loss of dependence on IGF-1 for proliferation of human thyroid adenoma cells.

@en

P2093

Williams DW

http://www.w3.org/2001/XMLSchema#string

Williams ED

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Wynford-Thomas D

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P2860

Dual control of cell growth by somatomedins and platelet-derived growth factor

Mitogenic proteins of pokeweed. I. Purification, characterization and mitogenic activity of two proteins from pokeweed (Phytolacca octandra).

Polarization of thyroid cells in culture: evidence for the basolateral localization of the iodide "pump" and of the thyroid-stimulating hormone receptor-adenyl cyclase complex.

Tissue concentrations of somatomedin C: further evidence for multiple sites of synthesis and paracrine or autocrine mechanisms of action.

Evolution of tumours and the impact of molecular oncology.

Location of epidermal growth factor receptors on porcine thyroid follicle cells and receptor regulation by thyrotropin.

Growth factor control of rat thyroid follicular cell proliferation.

Epidermal growth factor (EGF) and somatomedin C regulate G1 progression in competent BALB/c-3T3 cells.

Secretion of an insulin-like growth factor-I-related protein by human breast cancer cells.

Release of insulin-like growth factors and binding protein activity into serum-free medium of cultured human fibroblasts.

P2888

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535-539

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scholarly article

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10.1038/BJC.1988.124

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6

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57

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1014437576

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