Role of reactive oxygen species and gp91phox in endothelial dysfunction of pulmonary arteries induced by chronic hypoxia. - Wikidata - awgldk - TriplyDB

Role of reactive oxygen species and gp91phox in endothelial dysfunction of pulmonary arteries induced by chronic hypoxia.

Role of reactive oxygen species and gp91phox in endothelial dysfunction of pulmonary arteries induced by chronic hypoxia.

http://www.wikidata.org/entity/Q43054267

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Altered Nitric Oxide System in Cardiovascular and Renal Diseases DNA Damage and Pulmonary Hypertension A comprehensive review: the evolution of animal models in pulmonary hypertension research; are we there yet?NADPH oxidases in lung health and disease Impairment of NO-dependent relaxation in intralobar pulmonary arteries: comparison of urban particulate matter and manufactured nanoparticles Enhanced depolarization-induced pulmonary vasoconstriction following chronic hypoxia requires EGFR-dependent activation of NAD(P)H oxidase 2 Hydroethidine- and MitoSOX-derived red fluorescence is not a reliable indicator of intracellular superoxide formation: another inconvenient truth Pulmonary arterial hypertension: the clinical syndrome.NOX enzymes and pulmonary disease.Pulmonary endothelial cell NOX Regulation of NADPH oxidase in vascular endothelium: the role of phospholipases, protein kinases, and cytoskeletal proteins.Exposure of mice to chronic hypoxia attenuates pulmonary arterial contractile responses to acute hypoxia by increases in extracellular hydrogen peroxide Oxidative stress in fetal distress: potential prospects for diagnosis.PPAR{gamma} regulates hypoxia-induced Nox4 expression in human pulmonary artery smooth muscle cells through NF-{kappa}B.The Nox4 inhibitor GKT137831 attenuates hypoxia-induced pulmonary vascular cell proliferation NADPH oxidase 4 is expressed in pulmonary artery adventitia and contributes to hypertensive vascular remodeling Biopterin metabolism and eNOS expression during hypoxic pulmonary hypertension in mice.Pulmonary arterial responses to reactive oxygen species are altered in newborn piglets with chronic hypoxia-induced pulmonary hypertension The role of inflammation in hypoxic pulmonary hypertension: from cellular mechanisms to clinical phenotypes.Chronic hypoxia promotes pulmonary artery endothelial cell proliferation through H2O2-induced 5-lipoxygenase.Inhibition of MRP4 prevents and reverses pulmonary hypertension in mice.The proteome of Hypobaric Induced Hypoxic Lung: Insights from Temporal Proteomic Profiling for Biomarker Discovery NOX2 As a Target for Drug Development: Indications, Possible Complications, and Progress.Lung EC-SOD overexpression attenuates hypoxic induction of Egr-1 and chronic hypoxic pulmonary vascular remodeling.Functional characterization of voltage-dependent Ca(2+) channels in mouse pulmonary arterial smooth muscle cells: divergent effect of ROS Reactive oxygen species from NADPH oxidase contribute to altered pulmonary vascular responses in piglets with chronic hypoxia-induced pulmonary hypertension Attenuation of lipopolysaccharide-induced oxidative stress and apoptosis in fetal pulmonary artery endothelial cells by hypoxia.Protection of oral hydrogen water as an antioxidant on pulmonary hypertension The role of NADPH oxidase in chronic intermittent hypoxia-induced pulmonary hypertension in mice.Cardiac oxidative stress and remodeling following infarction: role of NADPH oxidase.Microparticulate/Nanoparticulate Powders of a Novel Nrf2 Activator and an Aerosol Performance Enhancer for Pulmonary Delivery Targeting the Lung Nrf2/Keap-1 Pathway.Reactive oxygen and nitrogen species in pulmonary hypertension.NADPH oxidases and reactive oxygen species at different stages of chronic hypoxia-induced pulmonary hypertension in newborn piglets.Reactive oxygen species in pulmonary vascular remodeling.Mechanisms of intermittent hypoxia induced hypertension.Differential roles of NADPH oxidases in vascular physiology and pathophysiology Lung oxidative damage by hypoxia Small things make a big difference: particulate matter and exercise.Reactive oxygen species as therapeutic targets in pulmonary hypertension.Circulating adhesion molecules in obstructive sleep apnea and cardiovascular disease.

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P2860

Role of reactive oxygen species and gp91phox in endothelial dysfunction of pulmonary arteries induced by chronic hypoxia.

http://www.wikidata.org/entity/Q43054267

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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2006年學術文章

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Role of reactive oxygen specie ...... es induced by chronic hypoxia.

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Role of reactive oxygen specie ...... es induced by chronic hypoxia.

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Role of reactive oxygen specie ...... es induced by chronic hypoxia.

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Role of reactive oxygen specie ...... es induced by chronic hypoxia.

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Role of reactive oxygen specie ...... es induced by chronic hypoxia.

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British Journal of Pharmacology

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Role of reactive oxygen specie ...... es induced by chronic hypoxia.

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P2093

Bernard Muller

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Fabrice Pourageaud

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Fleur Fresquet

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Jean-Pierre Savineau

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Roger Marthan

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Véronique Leblais

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P2860

Endothelial dysfunction in cardiovascular diseases: the role of oxidant stress

Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension

Soluble guanylate cyclase gene expression and localization in rat lung after exposure to hypoxia.

Evidence for dysregulation of dimethylarginine dimethylaminohydrolase I in chronic hypoxia-induced pulmonary hypertension.

Hydrogen peroxide is an endothelium-derived hyperpolarizing factor in mice.

Nitric oxide in the pathogenesis of vascular disease.

Role of nitric oxide in the pathogenesis of chronic pulmonary hypertension.

Endothelial dysfunction in the pulmonary vascular bed.

Role of alpha-adrenergic receptors in the effect of the beta-adrenergic receptor ligands, CGP 12177, bupranolol, and SR 59230A, on the contraction of rat intrapulmonary artery.

Endothelial dysfunction in pulmonary hypertension.

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5

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148

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Ralf P. Brandes

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7066866

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1751862

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