The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
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Potential of immunomodulatory agents as adjunct host-directed therapies for multidrug-resistant tuberculosisMacrophage arginase-1 controls bacterial growth and pathology in hypoxic tuberculosis granulomas.Human CD8+ T-cells recognizing peptides from Mycobacterium tuberculosis (Mtb) presented by HLA-E have an unorthodox Th2-like, multifunctional, Mtb inhibitory phenotype and represent a novel human T-cell subset.Biomarkers Can Identify Pulmonary Tuberculosis in HIV-infected Drug Users Months Prior to Clinical Diagnosis.Lack of the Transcription Factor Hypoxia-Inducible Factor 1α (HIF-1α) in Macrophages Accelerates the Necrosis of Mycobacterium avium-Induced Granulomas.A Mutation in IL4RA Is Associated with the Degree of Pathology in Human TB Patients.Mouse models of human TB pathology: roles in the analysis of necrosis and the development of host-directed therapies.Novel biomarker analysis of pleural effusion enhances differentiation of tuberculous from malignant pleural effusion.Neuron-specific enolase as a novel biomarker reflecting tuberculosis activity and treatment response.Type I IFN Inhibits Alternative Macrophage Activation during Mycobacterium tuberculosis Infection and Leads to Enhanced Protection in the Absence of IFN-γ Signaling.The bacillary and macrophage response to hypoxia in tuberculosis and the consequences for T cell antigen recognition.Metabolism via Arginase or Nitric Oxide Synthase: Two Competing Arginine Pathways in MacrophagesImmunometabolism within the tuberculosis granuloma: amino acids, hypoxia, and cellular respiration.Autophagy as a target for therapeutic uses of multifunctional peptides.Early Secreted Antigenic Target of 6 kDa of Mycobacterium tuberculosis Stimulates Macrophage Chemoattractant Protein-1 Production by Macrophages and Its Regulation by p38 Mitogen-Activated Protein Kinases and Interleukin-4.Cytokines and Chemokines in Mycobacterium tuberculosis Infection.Unravelling the networks dictating host resistance versus tolerance during pulmonary infections.Integrating Lung Physiology, Immunology, and Tuberculosis.Striking the right immunological balance prevents progression of tuberculosis.Type 2 immunity in tissue repair and fibrosis.l-Citrulline Metabolism in Mice Augments CD4+ T Cell Proliferation and Cytokine Production In Vitro, and Accumulation in the Mycobacteria-Infected Lung.Suppressor of Cytokine Signaling 3 in Macrophages Prevents Exacerbated Interleukin-6-Dependent Arginase-1 Activity and Early Permissiveness to Experimental Tuberculosis.Association of IL-13 rs20541 and rs1295686 variants with symptomatic asthma in a Saudi Arabian population.De Novo Fatty Acid Synthesis During Mycobacterial Infection Is a Prerequisite for the Function of Highly Proliferative T Cells, But Not for Dendritic Cells or Macrophages.Macrophage Heterogeneity in the Immunopathogenesis of Tuberculosis.
P2860
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P2860
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
@wuu
2014年学术文章
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2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
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name
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
@en
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
@nl
type
label
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
@en
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
@nl
prefLabel
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
@en
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
@nl
P2093
P2860
P356
P1476
The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.
@en
P2093
Andrew N J Mckenzie
Christoph Hölscher
Frank Brombacher
Hanna Erdmann
Jochen Behrends
Lisa Heitmann
Mahin Abad Dar
Stefan Ehlers
Tanja Schreiber
P2860
P304
P356
10.1002/PATH.4399
P577
2014-08-06T00:00:00Z