IL-17A-producing gammadelta T and Th17 lymphocytes mediate lung inflammation but not fibrosis in experimental silicosis. - Wikidata - awgldk - TriplyDB

IL-17A-producing gammadelta T and Th17 lymphocytes mediate lung inflammation but not fibrosis in experimental silicosis.

IL-17A-producing gammadelta T and Th17 lymphocytes mediate lung inflammation but not fibrosis in experimental silicosis.

http://www.wikidata.org/entity/Q43084852

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Oncoprotein mdig contributes to silica-induced pulmonary fibrosis by altering balance between Th17 and Treg T cells Silica, Silicosis, and Autoimmunity Endotoxin Exposure during Sensitization to Blomia tropicalis Allergens Shifts TH2 Immunity Towards a TH17-Mediated Airway Neutrophilic Inflammation: Role of TLR4 and TLR2 The Role of γδ T Cells in Fibrotic Diseases γδ T cells are required for pulmonary IL-17A expression after ozone exposure in mice: role of TNFα γδ T cells attenuate bleomycin-induced fibrosis through the production of CXCL10 Fibrogenic and redox-related but not proinflammatory genes are upregulated in Lewis rat model of chronic silicosis Uncoupling between inflammatory and fibrotic responses to silica: evidence from MyD88 knockout mice Blockade of Wnt/β-Catenin Pathway Aggravated Silica-Induced Lung Inflammation through Tregs Regulation on Th Immune Responses Overexpression of apolipoprotein A1 in the lung abrogates fibrosis in experimental silicosis Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis Baicalin Attenuates IL-17-Mediated Acetaminophen-Induced Liver Injury in a Mouse Model Neutrophil Extracellular Traps and Microcrystals Burn wound γδ T-cells support a Th2 and Th17 immune response Tregs promote the differentiation of Th17 cells in silica-induced lung fibrosis in mice.Significance of the balance between regulatory T (Treg) and T helper 17 (Th17) cells during hepatitis B virus related liver fibrosis Th17 can regulate silica-induced lung inflammation through an IL-1β-dependent mechanism Negative regulation of pulmonary Th17 responses by C3a anaphylatoxin during allergic inflammation in mice.Requirements for innate immune pathways in environmentally induced autoimmunity.Interleukin 13- and interleukin 17A-induced pulmonary hypertension phenotype due to inhalation of antigen and fine particles from air pollution Burn induces a Th-17 inflammatory response at the injury site.Epithelial transglutaminase 2 is needed for T cell interleukin-17 production and subsequent pulmonary inflammation and fibrosis in bleomycin-treated mice.Regulation and function of IL-17A- and IL-22-producing γδ T cells.Interleukin-22: immunobiology and pathology.Osteopontin Potentiates Pulmonary Inflammation and Fibrosis by Modulating IL-17/IFN-γ-secreting T-cell Ratios in Bleomycin-treated Mice Deletion of mTORC1 Activity in CD4+ T Cells Is Associated with Lung Fibrosis and Increased γδ T Cells.The proliferative effects of asbestos-exposed peripheral blood mononuclear cells on mesothelial cells.Role of IL-10-producing regulatory B cells in modulating T-helper cell immune responses during silica-induced lung inflammation and fibrosis.IL-17A-producing T cells are associated with the progression of lung adenocarcinoma Cytokine mediated tissue fibrosis Egr3 induces a Th17 response by promoting the development of γδ T cells.Vγ1⁺ γδT Cells Are Correlated With Increasing Expression of Eosinophil Cationic Protein and Metalloproteinase-7 in Chronic Rhinosinusitis With Nasal Polyps Inducing the Formation of Edema.Reduction of IL-17A might suppress the Th1 response and promote the Th2 response by boosting the function of Treg cells during silica-induced inflammatory response in vitro.Aerobiology and inhalation exposure to biological select agents and toxins.CD4+ T lymphocytes in lung fibrosis: diverse subsets, diverse functions.Understanding the origin, activation and regulation of matrix-producing myofibroblasts for treatment of fibrotic disease.Therapeutic opportunities of the IL-22-IL-22R1 system.Role of gamma-delta T cells in liver inflammation and fibrosis.The Jekyll and Hyde story of IL17-Producing γδT Cells.A mechanistic review of silica-induced inhalation toxicity.

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IL-17A-producing gammadelta T and Th17 lymphocytes mediate lung inflammation but not fibrosis in experimental silicosis.

http://www.wikidata.org/entity/Q43084852

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh-hant

name

IL-17A-producing gammadelta T ...... sis in experimental silicosis.

@en

IL-17A-producing gammadelta T ...... sis in experimental silicosis.

@nl

type

label

IL-17A-producing gammadelta T ...... sis in experimental silicosis.

@en

IL-17A-producing gammadelta T ...... sis in experimental silicosis.

@nl

prefLabel

IL-17A-producing gammadelta T ...... sis in experimental silicosis.

@en

IL-17A-producing gammadelta T ...... sis in experimental silicosis.

@nl

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JIMMUNOL.0900459

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Journal of Immunology

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IL-17A-producing gammadelta T ...... sis in experimental silicosis.

@en

P2093

Benoît Marien

http://www.w3.org/2001/XMLSchema#string

Catherine Uyttenhove

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Charlotte Van Vyve

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Francine Uwambayinema

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François Huaux

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Isabelle Couillin

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Jacques Van Snick

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Laure Dumoutier

http://www.w3.org/2001/XMLSchema#string

Sandra Lo Re

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Sybille van den Brûle

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6367-6377

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scholarly article

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10.4049/JIMMUNOL.0900459

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11

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184

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Jean-Christophe Renauld

Bernhard Ryffel

Isabelle Couillin

Dominique Lison

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2010-04-26T00:00:00Z

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43345846

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20421647

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