Clinical outcome after infection with Helicobacter pylori does not appear to be reliably predicted by the presence of any of the genes of the cag pathogenicity island.
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cagA Status and eradication treatment outcome of anti-Helicobacter pylori triple therapies in patients with nonulcer dyspepsiaComparative genomics of Helicobacter pylori isolates recovered from ulcer disease patients in England.A comprehensive sequence and disease correlation analyses for the C-terminal region of CagA protein of Helicobacter pylori.Prevalence of Helicobacter pylori vacA, cagA, iceA and oipA genotypes in Tunisian patientsAssociation of H pylori cagA and vacA genotypes and IL-8 gene polymorphisms with clinical outcome of infection in Iranian patients with gastrointestinal diseases.Determination of Helicobacter pylori virulence by simple gene analysis of the cag pathogenicity islandimplication of the structure of the Helicobacter pylori cag pathogenicity island in induction of interleukin-8 secretion.Role of Helicobacter pylori cag region genes in colonization and gastritis in two animal modelsEpidemiology and pattern of antibiotic resistance in Helicobacter pylori: scenario from Saudi ArabiaHelicobacter pylori in vegetables and salads: genotyping and antimicrobial resistance properties.Local and systemic immune and inflammatory responses to Helicobacter pylori strains.Helicobacter pylori-related diseases.The chips are down for Helicobacter pyloriIntact cag pathogenicity island of Helicobacter pylori without disease association in Kolkata, IndiaQuantitative evaluation of inflammatory and immune responses in the early stages of chronic Helicobacter pylori infection.Identification of cagA tyrosine phosphorylation DNA motifs in Helicobacter pylori isolates from peptic ulcer patients by novel PCR-restriction fragment length polymorphism and real-time fluorescence PCR assays.Conservation of the cag pathogenicity island is associated with vacA alleles and gastroduodenal disease in South African Helicobacter pylori isolatesGenetic diversity in the Helicobacter pylori cag pathogenicity island and effect on expression of anti-CagA serum antibody in UK patients with dyspepsiaHeterogeneity in the activity of Mexican Helicobacter pylori strains in gastric epithelial cells and its association with diversity in the cagA gene.Gene distribution of cagII in Helicobacter pylori-infected patients of Zhejiang Province.Serum positive cagA in patients with non-ulcer dyspepsia and peptic ulcer disease from two centers in different regions of Turkey.Distribution of cagG gene in Helicobacter pylori isolates from Chinese patients with different gastroduodenal diseases and its clinical and pathological significance.Expression of Helicobacter pylori virulence factors and associated expression profiles of inflammatory genes in the human gastric mucosaHelicobacter pylori cag pathogenicity island is associated with reduced expression of interleukin-4 (IL-4) mRNA and modulation of the IL-4delta2 mRNA isoform in human gastric mucosaCorrelation between cag pathogenicity island composition and Helicobacter pylori-associated gastroduodenal diseaseAssociation between cag-pathogenicity island in Helicobacter pylori isolates from peptic ulcer, gastric carcinoma, and non-ulcer dyspepsia subjects with histological changesAssociation of IS605 and cag-PAI of Helicobacter pylori Isolated from Patients with Gastrointestinal Diseases in Taiwan.The cag pathogenicity island of Helicobacter pylori is disrupted in the majority of patient isolates from different human populations.Distribution of open reading frames of plasticity region of strain J99 in Helicobacter pylori strains isolated from gastric carcinoma and gastritis patients in Costa Rica.Composition and gene expression of the cag pathogenicity island in Helicobacter pylori strains isolated from gastric carcinoma and gastritis patients in Costa RicaVirulence factors and antibiotic resistance of Helicobacter pylori isolated from raw milk and unpasteurized dairy products in IranHeterogeneity of cag genotypes in Helicobacter pylori isolates from human biopsy specimens.Simple method for determination of the number of Helicobacter pylori CagA variable-region EPIYA tyrosine phosphorylation motifs by PCRPrevalence of Helicobacter pylori vacA, cagA and iceA genotypes and correlation with clinical outcome.Diversity of Helicobacter pylori isolates in expression of antigens and induction of antibodies.Helicobacter pylori virulence factors in duodenal ulceration: A primary cause or a secondary infection causing chronicity.Chemokines and antimicrobial peptides have a cag-dependent early response to Helicobacter pylori infection in primary human gastric epithelial cells.Activation of Helicobacter pylori CagA by tyrosine phosphorylation is essential for dephosphorylation of host cell proteins in gastric epithelial cells.cag pathogenicity island of Helicobacter pylori in Korean children.Helicobacter pylori detection and genotyping in gastric biopsy specimens from Chennai patients (India).
P2860
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P2860
Clinical outcome after infection with Helicobacter pylori does not appear to be reliably predicted by the presence of any of the genes of the cag pathogenicity island.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Clinical outcome after infecti ...... the cag pathogenicity island.
@en
Clinical outcome after infecti ...... the cag pathogenicity island.
@nl
type
label
Clinical outcome after infecti ...... the cag pathogenicity island.
@en
Clinical outcome after infecti ...... the cag pathogenicity island.
@nl
prefLabel
Clinical outcome after infecti ...... the cag pathogenicity island.
@en
Clinical outcome after infecti ...... the cag pathogenicity island.
@nl
P2093
P2860
P356
P1433
P1476
Clinical outcome after infecti ...... the cag pathogenicity island.
@en
P2093
P2860
P304
P356
10.1136/GUT.43.6.752
P407
P577
1998-12-01T00:00:00Z