STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
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High Prevalence and Clinical Relevance of Genes Affected by Chromosomal Breaks in Colorectal Cancer.PS341 inhibits hepatocellular and colorectal cancer cells through the FOXO3/CTNNB1 signaling pathway.The Innate Immune Receptor NLRX1 Functions as a Tumor Suppressor by Reducing Colon Tumorigenesis and Key Tumor-Promoting Signals.Docosahexaenoic acid inhibits 12-O-tetradecanoylphorbol-13- acetate-induced fascin-1-dependent breast cancer cell migration by suppressing the PKCδ- and Wnt-1/β-catenin-mediated pathwaysDistinct signaling programs control human hematopoietic stem cell survival and proliferation.Inflammation and Colorectal Cancer.STAT3 exacerbates survival of cancer stem-like tumorspheres in EGFR-positive colorectal cancers: RNAseq analysis and therapeutic screening
P2860
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P2860
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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name
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
@en
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
@nl
type
label
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
@en
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
@nl
prefLabel
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
@en
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
@nl
P2093
P2860
P356
P1476
STAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.
@en
P2093
Abdolrahman S Nateri
Belal Muhammad
Darryl Jackson
Kanwal Baloch
Mohammad Ilyas
Saleh Al-Ghamdi
Salih Ibrahem
Wakkas Fadhil
P2860
P304
P356
10.1111/IEP.12102
P577
2014-10-28T00:00:00Z