Oxidative stress and modification of synaptic proteins in hippocampus after traumatic brain injury. - Wikidata - awgldk - TriplyDB

Oxidative stress and modification of synaptic proteins in hippocampus after traumatic brain injury.

Oxidative stress and modification of synaptic proteins in hippocampus after traumatic brain injury.

http://www.wikidata.org/entity/Q43104855

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Therapeutic potential of mood stabilizers lithium and valproic acid: beyond bipolar disorder Evaluating the Oxidative Stress in Inflammation: Role of Melatonin Bridge between neuroimmunity and traumatic brain injury Oxidative stress, DNA damage, and the telomeric complex as therapeutic targets in acute neurodegeneration Lesion Size Is Exacerbated in Hypoxic Rats Whereas Hypoxia-Inducible Factor-1 Alpha and Vascular Endothelial Growth Factor Increase in Injured Normoxic Rats: A Prospective Cohort Study of Secondary Hypoxia in Focal Traumatic Brain Injury Cortical hypoexcitation defines neuronal responses in the immediate aftermath of traumatic brain injury Traumatic brain injury and NADPH oxidase: a deep relationship Molecular mechanisms of cognitive dysfunction following traumatic brain injury Acute neuroimmune modulation attenuates the development of anxiety-like freezing behavior in an animal model of traumatic brain injury Therapies targeting lipid peroxidation in traumatic brain injury Effects of high fat diet on Morris maze performance, oxidative stress, and inflammation in rats: contributions of maternal diet Alzheimer’s disease and chronic traumatic encephalopathy: distinct but possibly overlapping disease entities S-glutathionylation: from molecular mechanisms to health outcomes Assessing neuro-systemic & behavioral components in the pathophysiology of blast-related brain injury.Alzheimer brain-derived amyloid β-protein impairs synaptic remodeling and memory consolidation.Age decreases macrophage IL-10 expression: Implications for functional recovery and tissue repair in spinal cord injury Probing astrocyte metabolism in vivo: proton magnetic resonance spectroscopy in the injured and aging brain Dietary curcumin supplementation counteracts reduction in levels of molecules involved in energy homeostasis after brain trauma Oxidative stress in the progression of Alzheimer disease in the frontal cortex.IGF-II promotes neuroprotection and neuroplasticity recovery in a long-lasting model of oxidative damage induced by glucocorticoids.Early mitochondrial dysfunction after cortical contusion injury The Effects of Blast Exposure on Protein Deimination in the Brain Role of low-level laser therapy in neurorehabilitation.Panel of synaptic protein ELISAs for evaluating neurological phenotype.Temporal and spatial dynamics of nrf2-antioxidant response elements mediated gene targets in cortex and hippocampus after controlled cortical impact traumatic brain injury in mice.NOX activity in brain aging: exacerbation by high fat diet.Oxidative stress in the developing brain: effects of postnatal glucocorticoid therapy and antioxidants in the rat.Effect of oxidative stress on homer scaffolding proteins Dicarboxylate carrier-mediated glutathione transport is essential for reactive oxygen species homeostasis and normal respiration in rat brain mitochondria.Lipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.Age-related mitochondrial changes after traumatic brain injury Critical role of NADPH oxidase in neuronal oxidative damage and microglia activation following traumatic brain injury.Experimental traumatic brain injury alters ethanol consumption and sensitivity.Neuroglobin-overexpression reduces traumatic brain lesion size in mice.Effects of diet and/or exercise in enhancing spinal cord sensorimotor learning.A systems biology strategy to identify molecular mechanisms of action and protein indicators of traumatic brain injury.Improved cognitive function after transcranial, light-emitting diode treatments in chronic, traumatic brain injury: two case reports.Synaptic change in the posterior cingulate gyrus in the progression of Alzheimer's disease Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice.Mitochondria-associated microRNAs in rat hippocampus following traumatic brain injury.

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Oxidative stress and modification of synaptic proteins in hippocampus after traumatic brain injury.

http://www.wikidata.org/entity/Q43104855

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2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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Oxidative stress and modificat ...... after traumatic brain injury.

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Oxidative stress and modificat ...... after traumatic brain injury.

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Oxidative stress and modificat ...... after traumatic brain injury.

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Oxidative stress and modificat ...... after traumatic brain injury.

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J.FREERADBIOMED.2008.04.038

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Free Radical Biology and Medicine

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Oxidative stress and modificat ...... after traumatic brain injury.

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Kelly N Roberts

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Mubeen A Ansari

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Stephen W Scheff

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P2860

Regulation of AMPA receptor GluR1 subunit surface expression by a 4. 1N-linked actin cytoskeletal association

Synapse-associated protein 97 selectively associates with a subset of AMPA receptors early in their biosynthetic pathway

Physiological role of mGSTA4-4, a glutathione S-transferase metabolizing 4-hydroxynonenal: generation and analysis of mGsta4 null mouse

Excitotoxic mechanisms and the role of astrocytic glutamate transporters in traumatic brain injury

GAP-43: an intrinsic determinant of neuronal development and plasticity

Laminins containing the beta2 chain modulate the precise organization of CNS synapses

The TRPM7 ion channel functions in cholinergic synaptic vesicles and affects transmitter release

Retrograde modulation of presynaptic release probability through signaling mediated by PSD-95-neuroligin

Expression of the prodynorphin gene after experimental brain injury and its role in behavioral dysfunction

Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

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