about
Munc18-1 expression levels control synapse recovery by regulating readily releasable pool sizeTau association with synaptic vesicles causes presynaptic dysfunction.Munc13-1 and Munc18-1 together prevent NSF-dependent de-priming of synaptic vesiclesEarly Golgi Abnormalities and Neurodegeneration upon Loss of Presynaptic Proteins Munc18-1, Syntaxin-1, or SNAP-25.Interactions Between SNAP-25 and Synaptotagmin-1 Are Involved in Vesicle Priming, Clamping Spontaneous and Stimulating Evoked Neurotransmission.The Sorting Receptor SorCS1 Regulates Trafficking of Neurexin and AMPA Receptors.Somatodendritic secretion in oxytocin neurons is upregulated during the female reproductive cycle.Phosphatidylinositol 4,5-bisphosphate optical uncaging potentiates exocytosis.Interdependence of PKC-dependent and PKC-independent pathways for presynaptic plasticity.Synaptogyrin-3 Mediates Presynaptic Dysfunction Induced by Tau.An Input-Specific Orphan Receptor GPR158-HSPG Interaction Organizes Hippocampal Mossy Fiber-CA3 SynapsesInnervation by a GABAergic Neuron Depresses Spontaneous Release in Glutamatergic Neurons and Unveils the Clamping Phenotype of Synaptotagmin-1A Modular Organization of LRR Protein-Mediated Synaptic Adhesion Defines Synapse IdentityGeneration of a human induced pluripotent stem cell-based model for tauopathies combining three microtubule-associated protein TAU mutations which displays several phenotypes linked to neurodegenerationSorCS1-mediated sorting in dendrites maintains neurexin axonal surface polarization required for synaptic functionSecreted amyloid-β precursor protein functions as a GABABR1a ligand to modulate synaptic transmissionXenotransplanted Human Cortical Neurons Reveal Species-Specific Development and Functional Integration into Mouse Visual CircuitsDystrophin deficiency leads to dysfunctional glutamate clearance in iPSC derived astrocytes
P50
Q28513229-C411D2C5-669E-4B47-990F-F9E8D500D15AQ33705540-BAA6554D-3873-422D-90D2-5F24A7BE162CQ33827873-C63D768F-D3DE-4246-81B6-26EFB71BF33EQ38872819-0D1E67C1-9640-4962-A0FE-2C0C9EA8B09FQ39157775-C282B9D8-D27B-479C-9E8D-DBC6871946F2Q43115262-C5C5D58C-7068-4204-AA07-C97FE788BACBQ44396232-DF8CBCF1-7913-4DAE-A941-C3FFF37AC4D3Q47160280-C2B13A2B-8C77-4756-BB9E-9E045A342931Q48198484-4A616112-C354-48F6-9347-9CA31EA4AFB2Q49028181-2C570CC4-F756-4C90-A1C3-8A9B9F005A67Q57071329-E0033813-BFD4-4AD7-8CC0-F24D68770146Q58361490-8D7C7D31-5070-434F-96C2-0EE2455621F6Q89462009-9EF49BE4-5E7C-4916-80C0-27B78DDDE3FCQ90415335-2B7697E4-F37B-4790-93B0-0452A046076FQ90970564-289CE031-8D24-4AC3-B13F-C252888A7006Q90988828-13853385-BD32-41FA-896D-4D6C786E03F7Q91422095-17CDAB1C-3B2B-46E0-A266-E2C9E965BF0CQ92756940-3998C8AD-1C14-4BC7-9CD7-D05CD5A2F303
P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Keimpe D Wierda
@en
Keimpe D Wierda
@es
Keimpe D Wierda
@sl
Keimpe Wierda
@nl
type
label
Keimpe D Wierda
@en
Keimpe D Wierda
@es
Keimpe D Wierda
@sl
Keimpe Wierda
@nl
altLabel
K. Wierda
@nl
K.D. Wierda
@nl
Keimpe D Wierda
@nl
prefLabel
Keimpe D Wierda
@en
Keimpe D Wierda
@es
Keimpe D Wierda
@sl
Keimpe Wierda
@nl
P108
P106
P108
P31
P496
0000-0002-8784-9490
P734
P735
P7449
PRS1337613