Suppression of Tie-1 in endothelial cells in vitro induces a change in the genome-wide expression profile reflecting an inflammatory function.
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XL-184, a MET, VEGFR-2 and RET kinase inhibitor for the treatment of thyroid cancer, glioblastoma multiforme and NSCLC.Tie1 controls angiopoietin function in vascular remodeling and inflammation.Using transcriptomics to identify and validate novel biomarkers of human skeletal muscle cancer cachexia.Gene expression profiling identifies inflammation and angiogenesis as distinguishing features of canine hemangiosarcoma.Tie1 attenuation reduces murine atherosclerosis in a dose-dependent and shear stress-specific mannerTie1 deletion inhibits tumor growth and improves angiopoietin antagonist therapyRole of Tie1 in shear stress and atherosclerosis.Tie-1: A potential target for anti-angiogenesis therapy.Tie1: an orphan receptor provides context for angiopoietin-2/Tie2 signaling.Therapeutic targeting of the angiopoietin-TIE pathway.Glycolytic cancer cells lacking 6-phosphogluconate dehydrogenase metabolize glucose to induce senescence.
P2860
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P2860
Suppression of Tie-1 in endothelial cells in vitro induces a change in the genome-wide expression profile reflecting an inflammatory function.
description
2009 nî lūn-bûn
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2009年の論文
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name
Suppression of Tie-1 in endoth ...... ting an inflammatory function.
@en
Suppression of Tie-1 in endoth ...... ting an inflammatory function.
@nl
type
label
Suppression of Tie-1 in endoth ...... ting an inflammatory function.
@en
Suppression of Tie-1 in endoth ...... ting an inflammatory function.
@nl
prefLabel
Suppression of Tie-1 in endoth ...... ting an inflammatory function.
@en
Suppression of Tie-1 in endoth ...... ting an inflammatory function.
@nl
P2860
P1433
P1476
Suppression of Tie-1 in endoth ...... ting an inflammatory function.
@en
P2093
Barden Chan
Vikas P Sukhatme
P2860
P304
P356
10.1016/J.FEBSLET.2009.02.027
P407
P577
2009-02-21T00:00:00Z