Glucose deprivation causes oxidative stress and stimulates aggresome formation and autophagy in cultured cardiac myocytes. - Wikidata - awgldk - TriplyDB

Glucose deprivation causes oxidative stress and stimulates aggresome formation and autophagy in cultured cardiac myocytes.

Glucose deprivation causes oxidative stress and stimulates aggresome formation and autophagy in cultured cardiac myocytes.

http://www.wikidata.org/entity/Q43153846

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Nutrient deprivation induces α-synuclein aggregation through endoplasmic reticulum stress response and SREBP2 pathway Interdependence of tetrapyrrole metabolism, the generation of oxidative stress and the mitigative oxidative stress response Inhibition of 6-phosphofructo-2-kinase (PFKFB3) induces autophagy as a survival mechanism Nrf2 deficiency exaggerates doxorubicin-induced cardiotoxicity and cardiac dysfunction.Mitochondrial fission is required for cardiomyocyte hypertrophy mediated by a Ca2+-calcineurin signaling pathway.2-Deoxy-D-glucose treatment of endothelial cells induces autophagy by reactive oxygen species-mediated activation of the AMP-activated protein kinase.Protective mechanisms of hypaconitine and glycyrrhetinic acid compatibility in oxygen and glucose deprivation injury.Dexamethasone-induced autophagy mediates muscle atrophy through mitochondrial clearance.Calcium and mitochondrial metabolism in ceramide-induced cardiomyocyte death.Autophagy and oxidative stress in cardiovascular diseases.Mitochondrial fission and autophagy in the normal and diseased heart Impairment of autophagic flux promotes glucose reperfusion-induced neuro2A cell death after glucose deprivation.Molecular mechanisms of autophagy in the cardiovascular system.The role of autophagy induced by tumor microenvironment in different cells and stages of cancer Endothelin-1 critically influences cardiac function via superoxide-MMP9 cascade.Nrf2 enhances myocardial clearance of toxic ubiquitinated proteins Cardiomyocyte death: mechanisms and translational implications Energy-preserving effects of IGF-1 antagonize starvation-induced cardiac autophagy Glycyrrhetinic acid protects H9c2 cells from oxygen glucose deprivation-induced injury through the PI3K/AKt signaling pathway.Quiescent fibroblasts are protected from proteasome inhibition-mediated toxicity Activation of SphK1 by K6PC-5 Inhibits Oxygen-Glucose Deprivation/Reoxygenation-Induced Myocardial Cell Death.Metformin Promotes Apoptosis but Suppresses Autophagy in Glucose-Deprived H4IIE Hepatocellular Carcinoma Cells.HERPUD1 protects against oxidative stress-induced apoptosis through downregulation of the inositol 1,4,5-trisphosphate receptor A kinase interacting protein (AKIP1) is a key regulator of cardiac stress.Glucose Modulation Induces Lysosome Formation and Increases Lysosomotropic Drug Sequestration via the P-Glycoprotein Drug Transporter.Oxidative stress and autophagy in cardiovascular homeostasis.Steroid receptor coactivator 1 is an integrator of glucose and NAD+/NADH homeostasis.Active autophagy in the tumor microenvironment: A novel mechanism for cancer metastasis.The wonders of 2-deoxy-D-glucose.Novel role of microRNAs in renal ischemia reperfusion injury.Autophagy and proteins involved in vesicular trafficking.T-type Ca2+ channel blocker, KYS05090 induces autophagy and apoptosis in A549 cells through inhibiting glucose uptake.Activation of Akt by SC79 protects myocardiocytes from oxygen and glucose deprivation (OGD)/re-oxygenation.Dynamic interplay between autophagic flux and Akt during melanoma progression in vitro.Characterization of early autophagy signaling by quantitative phosphoproteomics Mitochondrial fragmentation impairs insulin-dependent glucose uptake by modulating Akt activity through mitochondrial Ca2+ uptake.Roles for RAB24 in autophagy and disease.NAF-1 antagonizes starvation-induced autophagy through AMPK signaling pathway in cardiomyocytes.Regulation of autophagy by glucose in Mammalian cells.Effects of doxorubicin cancer therapy on autophagy and the ubiquitin-proteasome system in long-term cultured adult rat cardiomyocytes.

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P2860

Glucose deprivation causes oxidative stress and stimulates aggresome formation and autophagy in cultured cardiac myocytes.

http://www.wikidata.org/entity/Q43153846

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh-hant

name

Glucose deprivation causes oxi ...... in cultured cardiac myocytes.

@en

Glucose deprivation causes oxi ...... in cultured cardiac myocytes.

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type

label

Glucose deprivation causes oxi ...... in cultured cardiac myocytes.

@en

Glucose deprivation causes oxi ...... in cultured cardiac myocytes.

@nl

prefLabel

Glucose deprivation causes oxi ...... in cultured cardiac myocytes.

@en

Glucose deprivation causes oxi ...... in cultured cardiac myocytes.

@nl

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J.BBADIS.2010.02.002

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Biochimica et Biophysica Acta

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Glucose deprivation causes oxi ...... in cultured cardiac myocytes.

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Barbra Toro

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Carlos Sanhueza

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Clara Quiroga

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Daniela Munafo

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Guilermo Diaz-Araya

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Hugo Verdejo

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Jessica Díaz-Elizondo

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Lorena García

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María Isabel Colombo

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María-Julieta González

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509-518

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scholarly article

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10.1016/J.BBADIS.2010.02.002

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6

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Sergio Lavandero

Rodrigo Troncoso

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2010-02-20T00:00:00Z

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41531565

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20176105

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