Reactive oxygen species differently regulate renal tubular epithelial and interstitial cell proliferation after ischemia and reperfusion injury.
about
Omi/HtrA2 protease is associated with tubular cell apoptosis and fibrosis induced by unilateral ureteral obstructionAngiotensin II removes kidney resistance conferred by ischemic preconditioning.Loss of activator of G-protein signaling 3 impairs renal tubular regeneration following acute kidney injury in rodentsFolate receptor-targeted antioxidant therapy ameliorates renal ischemia-reperfusion injury.Regulation of MUTYH, a DNA Repair Enzyme, in Renal Proximal Tubular Epithelial Cells.Nox2 is a mediator of ischemia reperfusion injury.Unilateral nephrectomy elongates primary cilia in the remaining kidney via reactive oxygen species.Pharmacological inhibition of MyD88 homodimerization counteracts renal ischemia reperfusion-induced progressive renal injury in vivo and in vitro.Reduction of oxidative stress during recovery accelerates normalization of primary cilia length that is altered after ischemic injury in murine kidneys.Loss of poly(ADP-ribose) polymerase 1 attenuates renal fibrosis and inflammation during unilateral ureteral obstruction.Antioxidant properties of repaglinide and its protections against cyclosporine A-induced renal tubular injury.Hydrogen sulfide accelerates the recovery of kidney tubules after renal ischemia/reperfusion injury.Nrf2 deficiency promotes the progression from acute tubular damage to chronic renal fibrosis following unilateral ureteral obstruction.Recruitment and subsequent proliferation of bone marrow-derived cells in the postischemic kidney are important to the progression of fibrosis.Berberine protects HK-2 cells from hypoxia/reoxygenation induced apoptosis via inhibiting SPHK1 expression.Can Tissue Cilia Lengths and Urine Cilia Proteins Be Markers of Kidney Diseases?
P2860
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P2860
Reactive oxygen species differently regulate renal tubular epithelial and interstitial cell proliferation after ischemia and reperfusion injury.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Reactive oxygen species differ ...... chemia and reperfusion injury.
@en
Reactive oxygen species differ ...... chemia and reperfusion injury.
@nl
type
label
Reactive oxygen species differ ...... chemia and reperfusion injury.
@en
Reactive oxygen species differ ...... chemia and reperfusion injury.
@nl
prefLabel
Reactive oxygen species differ ...... chemia and reperfusion injury.
@en
Reactive oxygen species differ ...... chemia and reperfusion injury.
@nl
P1476
Reactive oxygen species differ ...... schemia and reperfusion injury
@en
P2093
Kwon Moo Park
Kyong-Jin Jung
P304
P356
10.1152/AJPRENAL.00701.2009
P50
P577
2010-02-17T00:00:00Z