Polycystins and mechanotransduction: From physiology to disease.
about
Continuous hydrostatic pressure induces differentiation phenomena in chondrocytes mediated by changes in polycystins, SOX9, and RUNX2.Fluid shear stress-induced TGF-β/ALK5 signaling in renal epithelial cells is modulated by MEK1/2.Primary cilia: Cell and molecular mechanosensors directing whole tissue function.Comprehensive transcriptome analysis of fluid shear stress altered gene expression in renal epithelial cells.Lengthening primary cilia enhances cellular mechanosensitivity.The role of transient receptor potential polycystin channels in bone diseases
P2860
Polycystins and mechanotransduction: From physiology to disease.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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name
Polycystins and mechanotransduction: From physiology to disease.
@en
Polycystins and mechanotransduction: From physiology to disease.
@nl
type
label
Polycystins and mechanotransduction: From physiology to disease.
@en
Polycystins and mechanotransduction: From physiology to disease.
@nl
prefLabel
Polycystins and mechanotransduction: From physiology to disease.
@en
Polycystins and mechanotransduction: From physiology to disease.
@nl
P2860
P356
P1476
Polycystins and mechanotransduction: From physiology to disease.
@en
P2093
Christina Piperi
Efthimia K Basdra
P2860
P304
P356
10.5493/WJEM.V5.I4.200
P577
2015-11-20T00:00:00Z