Gap junction remodeling and spironolactone-dependent reverse remodeling in the hypertrophied heart.
about
Cardiomyocyte protein trafficking: Relevance to heart disease and opportunities for therapeutic interventionPhenotyping of left and right ventricular function in mouse models of compensated hypertrophy and heart failure with cardiac MRIMechanoelectrical remodeling and arrhythmias during progression of hypertrophy.Phosphatase-resistant gap junctions inhibit pathological remodeling and prevent arrhythmias.Platelet TGF-β1 contributions to plasma TGF-β1, cardiac fibrosis, and systolic dysfunction in a mouse model of pressure overload.Impact of aldosterone antagonists on the substrate for atrial fibrillation: aldosterone promotes oxidative stress and atrial structural/electrical remodelingInhibition of angiotensin II-induced cardiac hypertrophy and associated ventricular arrhythmias by a p21 activated kinase 1 bioactive peptide.The Renin-Angiotensin system mediates the effects of stretch on conduction velocity, connexin43 expression, and redistribution in intact ventricle.Longitudinal arrhythmogenic remodelling in a mouse model of longstanding pressure overload.Super-resolution imaging reveals that loss of the C-terminus of connexin43 limits microtubule plus-end capture and NaV1.5 localization at the intercalated disc.Deciphering Arrhythmia Mechanisms - Tools of the TradeThe sarcoplasmic reticulum luminal thiol oxidase ERO1 regulates cardiomyocyte excitation-coupled calcium release and response to hemodynamic load.Lentivirus-mediated RNAi knockdown of the gap junction protein, Cx43, attenuates the development of vascular restenosis following balloon injury.GATA6 reporter gene reveals myocardial phenotypic heterogeneity that is related to variations in gap junction coupling.The connexin43 carboxyl terminus and cardiac gap junction organizationArrhythmogenic Remodeling in Murine Models of Deoxycorticosterone Acetate-Salt-Induced and 5/6-Subtotal Nephrectomy-Salt-Induced Cardiorenal DiseaseRemodeling of the peripheral cardiac conduction system in response to pressure overloadConnexin 43 is an emerging therapeutic target in ischemia/reperfusion injury, cardioprotection and neuroprotectionDesigner gap junctions that prevent cardiac arrhythmiasSpatial Heterogeneity of Cx43 is an Arrhythmogenic Substrate of Polymorphic Ventricular Tachycardias during Compensated Cardiac Hypertrophy in Rats.The cardiac connexome: Non-canonical functions of connexin43 and their role in cardiac arrhythmias.Regulation of cardiovascular connexins by mechanical forces and junctions.Remodeling of mechanical junctions and of microtubule-associated proteins accompany cardiac connexin43 lateralization.RU28318, an aldosterone antagonist, in combination with an ACE inhibitor and angiotensin receptor blocker attenuates cardiac dysfunction in diabetes.Managing the complexity of communication: regulation of gap junctions by post-translational modification.Specific Cx43 phosphorylation events regulate gap junction turnover in vivoCardiac fibrosis and arrhythmogenesis: the road to repair is paved with perils.Connexin43 cardiac gap junction remodeling: lessons from genetically engineered murine models.Mineralocorticoid receptor and cardiac arrhythmia.Arrhythmogenic cardiomyopathy and Brugada syndrome: diseases of the connexome.The role of extracellular matrix in age-related conduction disorders: a forgotten player?Mineralocorticoid receptor antagonists attenuate exaggerated exercise pressor reflex responses in hypertensive rats.Transmural gradients of myocardial structure and mechanics: Implications for fiber stress and strain in pressure overload.Spatio-temporal regulation of connexin43 phosphorylation and gap junction dynamics.Connexin 43 and Mitochondria in Cardiovascular Health and Disease.The noncanonical functions of Cx43 in the heart.Comparative evaluation of torasemide and spironolactone on adverse cardiac remodeling in a rat model of dilated cardiomyopathy.Connexin43 and the regulation of intercalated disc function.Reduction of fibrosis-related arrhythmias by chronic renin-angiotensin-aldosterone system inhibitors in an aged mouse model.Aldosterone Disrupts the Intercellular Flow of Glucose in Cardiac Muscle.
P2860
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P2860
Gap junction remodeling and spironolactone-dependent reverse remodeling in the hypertrophied heart.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Gap junction remodeling and sp ...... ng in the hypertrophied heart.
@en
Gap junction remodeling and sp ...... ng in the hypertrophied heart.
@nl
type
label
Gap junction remodeling and sp ...... ng in the hypertrophied heart.
@en
Gap junction remodeling and sp ...... ng in the hypertrophied heart.
@nl
prefLabel
Gap junction remodeling and sp ...... ng in the hypertrophied heart.
@en
Gap junction remodeling and sp ...... ng in the hypertrophied heart.
@nl
P2093
P2860
P1433
P1476
Gap junction remodeling and sp ...... ing in the hypertrophied heart
@en
P2093
Jiaxiang Qu
Lucrecia Márquez-Rosado
Luis I Garcia
Nefthi Sandeep
Paul D Lampe
P2860
P304
P356
10.1161/CIRCRESAHA.108.184044
P577
2008-12-18T00:00:00Z