Lack of MD-2 expression in human corneal epithelial cells is an underlying mechanism of lipopolysaccharide (LPS) unresponsiveness.
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Targeting toll-like receptor signaling as a novel approach to prevent ocular infectious diseasesTLR-Dependent Human Mucosal Epithelial Cell Responses to Microbial PathogensFlagellin-induced corneal antimicrobial peptide production and wound repair involve a novel NF-kappaB-independent and EGFR-dependent pathway.Role of Pattern Recognition Receptors in the Modulation of Antimicrobial Peptide Expression in the Corneal Epithelial Innate Response to F. solani.The role of toll-like receptor 4 in corneal epithelial wound healingInterferon-gamma-induced MD-2 protein expression and lipopolysaccharide (LPS) responsiveness in corneal epithelial cells is mediated by Janus tyrosine kinase-2 activation and direct binding of STAT1 protein to the MD-2 promoter.Inhibition of Corneal Inflammation by the Resolvin E1.Expression of toll-like receptor 4 contributes to corneal inflammation in experimental dry eye disease.Synergistic Effect of Dermatophagoides farinae and Lipopolysaccharides in Human Middle ear Epithelial Cells.Host defense at the ocular surfaceRadioiodination of an endotoxin·MD-2 complex generates a novel sensitive, high-affinity ligand for TLR4.Toll-like receptors in ocular surface disease.Toll-like receptors in ocular surface diseases: overview and new findings.Apple polysaccharide reduces NF-Kb mediated colitis-associated colon carcinogenesis.Exogenous MD-2 confers lipopolysaccharide responsiveness to human corneal epithelial cells with intracellular expression of TLR4 and CD14.Pretreatment with lipopolysaccharide modulates innate immunity in corneal fibroblasts challenged with Aspergillus fumigatus.The use of an IL-1 receptor antagonist peptide to control inflammation in the treatment of corneal limbal epithelial stem cell deficiency.When Clarity Is Crucial: Regulating Ocular Surface Immunity.Keratin 6a reorganization for ubiquitin-proteasomal processing is a direct antimicrobial response.Toll-like receptor-associated keratitis and strategies for its management.High-Mobility Group Box 1 in Dry Eye Inflammation.
P2860
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P2860
Lack of MD-2 expression in human corneal epithelial cells is an underlying mechanism of lipopolysaccharide (LPS) unresponsiveness.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
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2008年學術文章
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2008年學術文章
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name
Lack of MD-2 expression in hum ...... haride (LPS) unresponsiveness.
@en
Lack of MD-2 expression in hum ...... echanism of lipopolysaccharide
@nl
type
label
Lack of MD-2 expression in hum ...... haride (LPS) unresponsiveness.
@en
Lack of MD-2 expression in hum ...... echanism of lipopolysaccharide
@nl
prefLabel
Lack of MD-2 expression in hum ...... haride (LPS) unresponsiveness.
@en
Lack of MD-2 expression in hum ...... echanism of lipopolysaccharide
@nl
P2093
P2860
P356
P1476
Lack of MD-2 expression in hum ...... haride (LPS) unresponsiveness.
@en
P2093
Ashok Kumar
Fu-Shin X Yu
Jing Zhang
Michelle Wheater
P2860
P304
P356
10.1038/ICB.2008.75
P577
2008-10-21T00:00:00Z