JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
about
Multi-organ lesions in suckling mice infected with SARS-associated mammalian reovirus linked with apoptosis induced by viral proteins μ1 and σ1Independent regulation of reovirus membrane penetration and apoptosis by the mu1 phi domainBid regulates the pathogenesis of neurotropic reovirus.Isolation of reovirus T3D mutants capable of infecting human tumor cells independent of junction adhesion molecule-A.De novo assembly of Sockeye salmon kidney transcriptomes reveal a limited early response to piscine reovirus with or without infectious hematopoietic necrosis virus superinfection.Identification of Human Junctional Adhesion Molecule 1 as a Functional Receptor for the Hom-1 Calicivirus on Human Cells.From touchdown to transcription: the reovirus cell entry pathwayReovirus receptors, cell entry, and proapoptotic signaling.Reovirus activates a caspase-independent cell death pathway.Reovirus μ1 Protein Affects Infectivity by Altering Virus-Receptor Interactions.Conformational changes required for reovirus cell entry are sensitive to pH.Mammalian orthoreovirus T3D infects U-118 MG cell spheroids independent of junction adhesion molecule-A.Nonstructural protein σ1s mediates reovirus-induced cell cycle arrest and apoptosis.Apoptosis induced by mammalian reovirus is beta interferon (IFN) independent and enhanced by IFN regulatory factor 3- and NF-κB-dependent expression of Noxa.A proapoptotic peptide derived from reovirus outer capsid protein {micro}1 has membrane-destabilizing activity.A strategy for genetic modification of the spike-encoding segment of human reovirus T3D for reovirus targeting.Reovirus apoptosis and virulence are regulated by host cell membrane penetration efficiency.The reovirus μ1 340-343 loop controls entry related conformational changes.Reovirus outer capsid protein micro1 induces apoptosis and associates with lipid droplets, endoplasmic reticulum, and mitochondria.Viral gene expression potentiates reovirus-induced necrosis.In search of cathepsins: how reovirus enters host cells.IkappaB kinase subunits alpha and gamma are required for activation of NF-kappaB and induction of apoptosis by mammalian reovirus.Current understanding of reovirus oncolysis mechanisms.
P2860
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P2860
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
@zh-hans
2006年学术文章
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2006年學術文章
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name
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
@en
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
@nl
type
label
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
@en
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
@nl
prefLabel
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
@en
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
@nl
P2093
P2860
P1433
P1476
JAM-A-independent, antibody-mediated uptake of reovirus into cells leads to apoptosis.
@en
P2093
J Craig Forrest
Jacquelyn A Campbell
Mark W Hansberger
Terence S Dermody
P2860
P304
P356
10.1128/JVI.80.3.1261-1270.2006
P407
P577
2006-02-01T00:00:00Z