Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
about
Quantitative proteomic analysis reveals the perturbation of multiple cellular pathways in jurkat-T cells induced by doxorubicinSystems biology approaches to adverse drug effects: the example of cardio-oncology.Selenium attenuates adriamycin-induced cardiac dysfunction via restoring expression of ATP-sensitive potassium channels in rats.
P2860
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年學術文章
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name
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
@en
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
@nl
type
label
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
@en
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
@nl
prefLabel
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
@en
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
@nl
P2093
P2860
P1476
Measuring adriamycin-induced cardiac hemodynamic dysfunction with a proteomics approach.
@en
P2093
Cheng-Shi Piao
Do-Sung Kim
Geum-Hwa Lee
Hae-Kyung Kim
Han-Jung Chae
Hyung-Ryong Kim
Ki-Chan Ha
Seoung-Ju Park
Soo-Wan Chae
Wan-Hee Yoo
P2860
P304
P356
10.3109/08923970903440168
P577
2010-09-01T00:00:00Z