Interleukin-1 receptor signaling protects mice from lethal intestinal damage caused by the attaching and effacing pathogen Citrobacter rodentium. - Wikidata - awgldk - TriplyDB

Interleukin-1 receptor signaling protects mice from lethal intestinal damage caused by the attaching and effacing pathogen Citrobacter rodentium.

Interleukin-1 receptor signaling protects mice from lethal intestinal damage caused by the attaching and effacing pathogen Citrobacter rodentium.

http://www.wikidata.org/entity/Q43192584

about

Immunomodulatory functions of type I interferons Inflammasome in intestinal inflammation and cancer Toll-like receptors in pathophysiology of liver diseases HVEM signalling at mucosal barriers provides host defence against pathogenic bacteria Caspase-1 activation via Rho GTPases: a common theme in mucosal infections?MyD88 signaling in dendritic cells and the intestinal epithelium controls immunity against intestinal infection with C. rodentium.Multivariate modeling identifies neutrophil- and Th17-related factors as differential serum biomarkers of chronic murine colitis.NOD-like receptors in intestinal homeostasis and epithelial tissue repair.Kinetic approach to pathway attenuation using XOMA 052, a regulatory therapeutic antibody that modulates interleukin-1beta activity.Altering host resistance to infections through microbial transplantation.Deregulated inflammasome signaling in disease.The NLRP3 inflammasome protects against loss of epithelial integrity and mortality during experimental colitis.Caspase-11 is expressed in the colonic mucosa and protects against dextran sodium sulfate-induced colitis Interleukin 1 receptor signaling regulates DUBA expression and facilitates Toll-like receptor 9-driven antiinflammatory cytokine production.Interleukin-1 receptor signaling is required to overcome the effects of pertussis toxin and for efficient infection- or vaccination-induced immunity against Bordetella pertussis.The Nlrp3 inflammasome: contributions to intestinal homeostasis.Epithelial phosphatidylinositol-3-kinase signaling is required for β-catenin activation and host defense against Citrobacter rodentium infection.SIGIRR, a negative regulator of TLR/IL-1R signalling promotes Microbiota dependent resistance to colonization by enteric bacterial pathogens.A balanced IL-1β activity is required for host response to Citrobacter rodentium infection.Selective role for tumor necrosis factor-α, but not interleukin-1 or Kupffer cells, in down-regulation of CYP3A11 and CYP3A25 in livers of mice infected with a noninvasive intestinal pathogen.Distinct roles for Nod2 protein and autocrine interleukin-1beta in muramyl dipeptide-induced mitogen-activated protein kinase activation and cytokine secretion in human macrophages SIGNR3-dependent immune regulation by Lactobacillus acidophilus surface layer protein A in colitis.NF-κB in the regulation of epithelial homeostasis and inflammation.Host innate recognition of an intestinal bacterial pathogen induces TRIF-dependent protective immunity.The epithelial danger signal IL-1α is a potent activator of fibroblasts and reactivator of intestinal inflammation.The role of innate immunity in the host defense against intestinal bacterial pathogens.Interleukin-1 (IL-1) signaling in intestinal stromal cells controls KC/ CXCL1 secretion, which correlates with recruitment of IL-22- secreting neutrophils at early stages of Citrobacter rodentium infection.Functions of innate immune cells and commensal bacteria in gut homeostasis.The role of phosphoinositide 3-kinase signaling in intestinal inflammation Generation of a Novel T Cell Specific Interleukin-1 Receptor Type 1 Conditional Knock Out Mouse Reveals Intrinsic Defects in Survival, Expansion and Cytokine Production of CD4 T Cells.HVEM: An unusual TNF receptor family member important for mucosal innate immune responses to microbes.Precarious balance: Th17 cells in host defense.An innately dangerous balancing act: intestinal homeostasis, inflammation, and colitis-associated cancer.Innate host responses to enteric bacterial pathogens: a balancing act between resistance and tolerance.Inflammasomes in carcinogenesis and anticancer immune responses.Interactions between Nod-Like Receptors and Intestinal Bacteria.Role of inflammasomes in intestinal inflammation and Crohn's disease.Protective and detrimental roles of inflammasomes in disease.IL-1RT1 signaling antagonizes IL-11 induced STAT3 dependent cardiac and antral stomach tumor development through myeloid cell enrichment.Pre-treatment with IL-1β enhances the efficacy of MSC transplantation in DSS-induced colitis.

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Interleukin-1 receptor signaling protects mice from lethal intestinal damage caused by the attaching and effacing pathogen Citrobacter rodentium.

http://www.wikidata.org/entity/Q43192584

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

@zh-cn

2008年学术文章

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2008年学术文章

@zh-my

2008年学术文章

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2008年學術文章

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2008年學術文章

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name

Interleukin-1 receptor signali ...... athogen Citrobacter rodentium.

@en

Interleukin-1 receptor signali ...... athogen Citrobacter rodentium.

@nl

type

label

Interleukin-1 receptor signali ...... athogen Citrobacter rodentium.

@en

Interleukin-1 receptor signali ...... athogen Citrobacter rodentium.

@nl

prefLabel

Interleukin-1 receptor signali ...... athogen Citrobacter rodentium.

@en

Interleukin-1 receptor signali ...... athogen Citrobacter rodentium.

@nl

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Infection and Immunity

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Interleukin-1 receptor signali ...... pathogen Citrobacter rodentium

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P2093

Daniel Kalman

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Didier Merlin

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Melanie A Sherman

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Sarah L Lebeis

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P2860

Invasive Escherichia coli are a feature of Crohn's disease

Cytosolic extract induces Tir translocation and pedestals in EPEC-infected red blood cells

Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases

Recognition of commensal microflora by toll-like receptors is required for intestinal homeostasis

Food-related illness and death in the United States

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

Biologic basis for interleukin-1 in disease

Activated macrophages are an adaptive element of the colonic epithelial progenitor niche necessary for regenerative responses to injury.

Effect of prior experimental human enteropathogenic Escherichia coli infection on illness following homologous and heterologous rechallenge.

Mast cells limit systemic bacterial dissemination but not colitis in response to Citrobacter rodentium

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604-614

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10.1128/IAI.00907-08

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2

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77

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Kimberly R Powell

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23655921

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19075023

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2632048

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