about
Phagocytes as Corrupted Policemen in Cancer-Related InflammationPTX3 is an extrinsic oncosuppressor regulating complement-dependent inflammation in cancer.Role of Toll interleukin-1 receptor (IL-1R) 8, a negative regulator of IL-1R/Toll-like receptor signaling, in resistance to acute Pseudomonas aeruginosa lung infection.Decoys and Regulatory "Receptors" of the IL-1/Toll-Like Receptor Superfamily.The long pentraxin PTX3 at the crossroads between innate immunity and tissue remodelling.Tumor associated macrophages and neutrophils in cancer.Negative regulatory receptors of the IL-1 family.The long pentraxin PTX3 as a key component of humoral innate immunity and a candidate diagnostic for inflammatory diseases.The humoral pattern recognition molecule PTX3 is a key component of innate immunity against urinary tract infection.Occurrence and significance of tumor-associated neutrophils in patients with colorectal cancer.TIR8/SIGIRR is an Interleukin-1 Receptor/Toll Like Receptor Family Member with Regulatory Functions in Inflammation and ImmunityPTX3 acts as an extrinsic oncosuppressorThe activation of liver X receptors inhibits toll-like receptor-9-induced foam cell formation.IL-1R8 is a checkpoint in NK cells regulating anti-tumour and anti-viral activity.Resolving the dark side of therapy-driven cancer cell death.NK Cells Stimulate Recruitment of cDC1 into the Tumor Microenvironment Promoting Cancer Immune Control.Plasmacytoid Dendritic Cells Alter the Antitumor Activity of CpG-Oligodeoxynucleotides in a Mouse Model of Lung Carcinoma
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Eduardo Bonavita
@ast
Eduardo Bonavita
@en
Eduardo Bonavita
@es
Eduardo Bonavita
@nl
Eduardo Bonavita
@sl
type
label
Eduardo Bonavita
@ast
Eduardo Bonavita
@en
Eduardo Bonavita
@es
Eduardo Bonavita
@nl
Eduardo Bonavita
@sl
prefLabel
Eduardo Bonavita
@ast
Eduardo Bonavita
@en
Eduardo Bonavita
@es
Eduardo Bonavita
@nl
Eduardo Bonavita
@sl
P1053
P-4370-2014
P106
P1153
35809920800
P21
P2798
P31
P3829
P496
0000-0003-2335-5827