Histone deacetylase 3 is critical in endothelial survival and atherosclerosis development in response to disturbed flow. - Wikidata - awgldk - TriplyDB

Histone deacetylase 3 is critical in endothelial survival and atherosclerosis development in response to disturbed flow.

Histone deacetylase 3 is critical in endothelial survival and atherosclerosis development in response to disturbed flow.

http://www.wikidata.org/entity/Q43212310

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Unspliced X-box-binding protein 1 (XBP1) protects endothelial cells from oxidative stress through interaction with histone deacetylase 3 Histone Deacetylases and Cardiometabolic Diseases Endothelial epigenetics in biomechanical stress: disturbed flow-mediated epigenomic plasticity in vivo and in vitro Epigenetic regulation of endothelial-cell-mediated vascular repair Targeting histone deacetylases: perspectives for epigenetic-based therapy in cardio-cerebrovascular disease The role of epigenetics in the endothelial cell shear stress response and atherosclerosis Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives HDAC3 acts as a negative regulator of angiogenesis.Atherogenic factors and their epigenetic relationships Beyond histone and deacetylase: an overview of cytoplasmic histone deacetylases and their nonhistone substrates Role of histone deacetylases in transcription factor regulation and cell cycle modulation in endothelial cells in response to disturbed flow.Aspirin-induced histone acetylation in endothelial cells enhances synthesis of the secreted isoform of netrin-1 thus inhibiting monocyte vascular infiltration.Force-specific activation of Smad1/5 regulates vascular endothelial cell cycle progression in response to disturbed flow.Next steps in cardiovascular disease genomic research--sequencing, epigenetics, and transcriptomics Histone deacetylase 3 unconventional splicing mediates endothelial-to-mesenchymal transition through transforming growth factor β2.Flow-induced HDAC1 phosphorylation and nuclear export in angiogenic sprouting.Mechanical regulation of epigenetics in vascular biology and pathobiology Hyaluronic acid promotes angiogenesis by inducing RHAMM-TGFβ receptor interaction via CD44-PKCδ.Adenosine monophosphate-activated protein kinase-α2 deficiency promotes vascular smooth muscle cell migration via S-phase kinase-associated protein 2 upregulation and E-cadherin downregulation.Protein post-translational modifications and regulation of pluripotency in human stem cells.Role of histone deacetylases in vascular cell homeostasis and arteriosclerosis.Molecular biology of atherosclerosis.Endothelial heterogeneity in the umbilico-placental unit: DNA methylation as an innuendo of epigenetic diversity.Shear stress-initiated signaling and its regulation of endothelial function.Epigenetic reprogramming in atherosclerosis.Biofluids, cell mechanics and epigenetics: Flow-induced epigenetic mechanisms of endothelial gene expression.Epigenetics in the Vascular Endothelium: Looking From a Different Perspective in the Epigenomics Era.Translational Perspective on Epigenetics in Cardiovascular Disease.Histone deacetylase-high mobility group box-1 pathway targeted by hypaconitine suppresses the apoptosis of endothelial cells.Epigenetic Regulation of Monocyte and Macrophage Function.Epigenetic Changes in Diabetes and Cardiovascular Risk.Effects of shear stress on endothelial cells: go with the flow.Targeting macrophage Histone deacetylase 3 stabilizes atherosclerotic lesions.Galectin-9 protein expression in endothelial cells is positively regulated by histone deacetylase 3.CRISPR/Cas9 editing reveals novel mechanisms of clustered microRNA regulation and function.Epigenetics and trained immunity.MicroRNA-10a is crucial for endothelial response to different flow patterns via interaction of retinoid acid receptors and histone deacetylases.Transcriptional regulation of Nox4 by histone deacetylases in human endothelial cells.Histone deacetylase 3 is associated with gastric cancer cell growth via the miR-454-mediated targeting of CHD5.Epigenetics in diabetic nephropathy, immunity and metabolism.

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Histone deacetylase 3 is critical in endothelial survival and atherosclerosis development in response to disturbed flow.

http://www.wikidata.org/entity/Q43212310

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

Histone deacetylase 3 is criti ...... in response to disturbed flow.

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Histone deacetylase 3 is criti ...... in response to disturbed flow.

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type

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Histone deacetylase 3 is criti ...... in response to disturbed flow.

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Histone deacetylase 3 is criti ...... in response to disturbed flow.

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Histone deacetylase 3 is criti ...... in response to disturbed flow.

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Histone deacetylase 3 is criti ...... in response to disturbed flow.

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CIRCULATIONAHA.109.890491

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Histone deacetylase 3 is criti ...... in response to disturbed flow.

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Andriana Margariti

http://www.w3.org/2001/XMLSchema#string

Anna Elena Pepe

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Anna Zampetaki

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Elena deFalco

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Gang Wang

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Gillian Cockerill

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Hongling Li

http://www.w3.org/2001/XMLSchema#string

Justin C Mason

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Lingfang Zeng

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Ouassila Habi

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132-142

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scholarly article

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10.1161/CIRCULATIONAHA.109.890491

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1

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121

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2009-12-21T00:00:00Z

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20026773

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atherosclerosis