Reduced SERCA2 abundance decreases the propensity for Ca2+ wave development in ventricular myocytes.
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Chronic lead exposure increases blood pressure and myocardial contractility in ratsCa²⁺-dependent phosphorylation of Ca²⁺ cycling proteins generates robust rhythmic local Ca²⁺ releases in cardiac pacemaker cells.Deregulated Ca2+ cycling underlies the development of arrhythmia and heart disease due to mutant obscurinTargeting intracellular calcium cycling in catecholaminergic polymorphic ventricular tachycardia: a theoretical investigation.Calcium dynamics in the ventricular myocytes of SERCA2 knockout mice: A modeling studyPerturbed atrial calcium handling in an ovine model of heart failure: potential roles for reductions in the L-type calcium current.Prominent heart organ-level performance deficits in a genetic model of targeted severe and progressive SERCA2 deficiency.Substrate stiffness increases twitch power of neonatal cardiomyocytes in correlation with changes in myofibril structure and intracellular calciumTargeting cardiomyocyte Ca2+ homeostasis in heart failure.Instabilities of the resting state in a mathematical model of calcium handling in cardiac myocytes.Calcium alternans in a couplon network model of ventricular myocytes: role of sarcoplasmic reticulum load.Slow Calcium-Depolarization-Calcium waves may initiate fast local depolarization waves in ventricular tissue.Synchrony of cardiomyocyte Ca(2+) release is controlled by T-tubule organization, SR Ca(2+) content, and ryanodine receptor Ca(2+) sensitivity.Ca(2+)-Clock-Dependent Pacemaking in the Sinus Node Is Impaired in Mice with a Cardiac Specific Reduction in SERCA2 AbundanceProtein Inhibitor of NOS1 Plays a Central Role in the Regulation of NOS1 Activity in Human Dilated Hearts.Cardiac fibrosis in mice expressing an inducible myocardial-specific Cre driver.No rest for the weary: diastolic calcium homeostasis in the normal and failing myocardium.The sarcoplasmic reticulum and the evolution of the vertebrate heart.Cardiomyocyte Ca2+ dynamics: clinical perspectives.Cab45S promotes cell proliferation through SERCA2b inhibition and Ca2+ signaling.Exercise training prevents ventricular tachycardia in CPVT1 due to reduced CaMKII-dependent arrhythmogenic Ca2+ release.Mitochondrial pathways to cardiac recovery: TFAM.Facilitation of cytosolic calcium wave propagation by local calcium uptake into the sarcoplasmic reticulum in cardiac myocytes.Long-term levosimendan treatment improves systolic function and myocardial relaxation in mice with cardiomyocyte-specific disruption of the Serca2 gene.Phospholamban ablation rescues the enhanced propensity to arrhythmias of mice with CaMKII-constitutive phosphorylation of RyR2 at site S2814.Impaired left ventricular mechanical and energetic function in mice after cardiomyocyte-specific excision of Serca2.Ca(2+) wave probability is determined by the balance between SERCA2-dependent Ca(2+) reuptake and threshold SR Ca(2+) content.
P2860
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P2860
Reduced SERCA2 abundance decreases the propensity for Ca2+ wave development in ventricular myocytes.
description
2009 nî lūn-bûn
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2009年の論文
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2009年学术文章
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2009年学术文章
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2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
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2009年学术文章
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2009年學術文章
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name
Reduced SERCA2 abundance decre ...... pment in ventricular myocytes.
@en
Reduced SERCA2 abundance decre ...... pment in ventricular myocytes.
@nl
type
label
Reduced SERCA2 abundance decre ...... pment in ventricular myocytes.
@en
Reduced SERCA2 abundance decre ...... pment in ventricular myocytes.
@nl
prefLabel
Reduced SERCA2 abundance decre ...... pment in ventricular myocytes.
@en
Reduced SERCA2 abundance decre ...... pment in ventricular myocytes.
@nl
P2093
P2860
P50
P356
P1476
Reduced SERCA2 abundance decre ...... pment in ventricular myocytes.
@en
P2093
Karina Hougen
Kristin B Andersson
Mathis K Stokke
Ulla H Enger
William E Louch
P2860
P356
10.1093/CVR/CVP401
P577
2009-12-17T00:00:00Z