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Impact of Glucocorticoid Excess on Glucose Tolerance: Clinical and Preclinical EvidenceImplications of palmitoleic acid (palmitoleate) on glucose homeostasis, insulin resistance and diabetes.The cannabinoid ligand LH-21 reduces anxiety and improves glucose handling in diet-induced obese pre-diabetic mice.Pancreatic alpha-cell dysfunction contributes to the disruption of glucose homeostasis and compensatory insulin hypersecretion in glucocorticoid-treated rats.Glucocorticoid treatment and endocrine pancreas function: implications for glucose homeostasis, insulin resistance and diabetes.Impact of Fish Oil Supplementation and Interruption of Fructose Ingestion on Glucose and Lipid Homeostasis of Rats Drinking Different Concentrations of FructoseLiraglutide counteracts obesity and glucose intolerance in a mouse model of glucocorticoid-induced metabolic syndrome.Augmented β-Cell Function and Mass in Glucocorticoid-Treated Rodents Are Associated with Increased Islet Ir-β /AKT/mTOR and Decreased AMPK/ACC and AS160 Signaling.Leucine supplementation augments insulin secretion in pancreatic islets of malnourished mice.Reduced insulin secretion in protein malnourished mice is associated with multiple changes in the beta-cell stimulus-secretion coupling.The adaptive compensations in endocrine pancreas from glucocorticoid-treated rats are reversible after the interruption of treatment.Exercise training prevents hyperinsulinemia, muscular glycogen loss and muscle atrophy induced by dexamethasone treatment.Involvement of the cholinergic pathway in glucocorticoid-induced hyperinsulinemia in rats.Augmentation of insulin secretion by leucine supplementation in malnourished rats: possible involvement of the phosphatidylinositol 3-phosphate kinase/mammalian target protein of rapamycin pathway.Glucocorticoids in vivo induce both insulin hypersecretion and enhanced glucose sensitivity of stimulus-secretion coupling in isolated rat islets.Reduced pancreatic beta-cell mass is associated with decreased FoxO1 and Erk1/2 protein phosphorylation in low-protein malnourished rats.Evaluation of lipid homeostasis in the late gestational period of rats exposed to dexamethasone.Cortistatin hyperpolarizes pancreatic beta cell membrane and reduces glucose-stimulated insulin secretion.High doses of dexamethasone induce increased beta-cell proliferation in pancreatic rat islets.Reduced glucose-induced insulin secretion in low-protein-fed rats is associated with altered pancreatic islets redox status.Glucose intolerance induced by glucocorticoid excess is further impaired by co-administration with β-hydroxy-β-methylbutyrate in rats.Dexamethasone treatment in vivo counteracts the functional pancreatic islet alterations caused by malnourishment in rats.Hyperinsulinemia caused by dexamethasone treatment is associated with reduced insulin clearance and lower hepatic activity of insulin-degrading enzyme.INGAP-PP up-regulates the expression of genes and proteins related to K+ ATP channels and ameliorates Ca2+ handling in cultured adult rat islets.Functional alterations in endocrine pancreas of rats with different degrees of dexamethasone-induced insulin resistance.Glucose homeostasis in two degrees of sepsis lethality induced by caecum ligation and puncture in mice.Tissue integrity, costs and time associated with different agents for histological bone preparation.Glucose homeostasis in rats treated with 4-vinylcyclohexene diepoxide is not worsened by dexamethasone treatment.Age- and gender-related changes in glucose homeostasis in glucocorticoid-treated rats.Fish oil supplementation attenuates changes in plasma lipids caused by dexamethasone treatment in rats.Functional and structural adaptations in the pancreatic α-cell and changes in glucagon signaling during protein malnutrition.Disruption of glucose tolerance caused by glucocorticoid excess in rats is partially prevented, but not attenuated, by arjunolic acid.High-fat diet obesity associated with insulin resistance increases cell proliferation, estrogen receptor, and PI3K proteins in rat ventral prostate.Increased pancreatic islet mass is accompanied by activation of the insulin receptor substrate-2/serine-threonine kinase pathway and augmented cyclin D2 protein levels in insulin-resistant rats.A high-fat diet fed during different periods of life impairs steroidogenesis of rat Leydig cells.JNK and IKKβ phosphorylation is reduced by glucocorticoids in adipose tissue from insulin-resistant rats.A role for the putative cannabinoid receptor GPR55 in the islets of Langerhans.Prevention of Elevation in Plasma Triacylglycerol with High-Dose Bezafibrate Treatment Abolishes Insulin Resistance and Attenuates Glucose Intolerance Induced by Short-Term Treatment with Dexamethasone in RatsGlucose Homeostasis Is Not Affected in a Murine Model of Parkinson's Disease Induced by 6-OHDADexamethasone during pregnancy impairs maternal pancreatic β-cell renewal during lactation
P50
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P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Alex Rafacho
@ast
Alex Rafacho
@en
Alex Rafacho
@es
Alex Rafacho
@nl
type
label
Alex Rafacho
@ast
Alex Rafacho
@en
Alex Rafacho
@es
Alex Rafacho
@nl
prefLabel
Alex Rafacho
@ast
Alex Rafacho
@en
Alex Rafacho
@es
Alex Rafacho
@nl
P1053
O-8609-2016
P106
P21
P31
P3829
P496
0000-0002-8637-6097