Morphine prevents the mitochondrial permeability transition pore opening through NO/cGMP/PKG/Zn2+/GSK-3beta signal pathway in cardiomyocytes.
about
Biochemistry of mobile zinc and nitric oxide revealed by fluorescent sensors.Overexpression of Ubiquinol-Cytochrome c Reductase Core Protein 1 May Protect H9c2 Cardiac Cells by Binding with Zinc.Morphine induces preconditioning via activation of mitochondrial K(Ca) channelsEffect of remifentanil on mitochondrial oxygen consumption of cultured human hepatocytes.Imaging of reactive oxygen species burst from mitochondria using laser scanning confocal microscopy.Morphine-induced postconditioning modulates mitochondrial permeability transition pore opening via delta-1 opioid receptors activation in isolated rat hearts.Glycogen synthase kinase-3α limits ischemic injury, cardiac rupture, post-myocardial infarction remodeling and death.Astragaloside IV inhibits oxidative stress-induced mitochondrial permeability transition pore opening by inactivating GSK-3β via nitric oxide in H9c2 cardiac cells.Soluble guanylate cyclase activation during ischemic injury in mice protects against postischemic inflammation at the mitochondrial level.Tauroursodeoxycholic acid inhibits endoplasmic reticulum stress, blocks mitochondrial permeability transition pore opening, and suppresses reperfusion injury through GSK-3ß in cardiac H9c2 cells.Myocardial ischemic post-conditioning protects the lung against myocardial ischemia/reperfusion-induced damage by activating GSK-3β.Research review on the pharmacological effects of astragaloside IV.Interplay Between Cytosolic Free Zn2+ and Mitochondrion Morphological Changes in Rat Ventricular Cardiomyocytes.Zn2+ and mPTP Mediate Endoplasmic Reticulum Stress Inhibition-Induced Cardioprotection Against Myocardial Ischemia/Reperfusion Injury.Intracellular Zn(2+) Increase in Cardiomyocytes Induces both Electrical and Mechanical Dysfunction in Heart via Endogenous Generation of Reactive Nitrogen Species.The role of glycogen synthase kinase 3 beta in brain injury induced by myocardial ischemia/reperfusion injury in a rat model of diabetes mellitus.Roles of Endoplasmic Reticulum Stress in NECA-Induced Cardioprotection against Ischemia/Reperfusion Injury.Myocardial Opioid Receptors in Conditioning and Cytoprotection.
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P2860
Morphine prevents the mitochondrial permeability transition pore opening through NO/cGMP/PKG/Zn2+/GSK-3beta signal pathway in cardiomyocytes.
description
2009 nî lūn-bûn
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2009年の論文
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2009年学术文章
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2009年学术文章
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2009年学术文章
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2009年学术文章
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2009年学术文章
@zh-my
2009年学术文章
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2009年學術文章
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name
Morphine prevents the mitochon ...... nal pathway in cardiomyocytes.
@en
Morphine prevents the mitochon ...... nal pathway in cardiomyocytes.
@nl
type
label
Morphine prevents the mitochon ...... nal pathway in cardiomyocytes.
@en
Morphine prevents the mitochon ...... nal pathway in cardiomyocytes.
@nl
prefLabel
Morphine prevents the mitochon ...... nal pathway in cardiomyocytes.
@en
Morphine prevents the mitochon ...... nal pathway in cardiomyocytes.
@nl
P2093
P2860
P1476
Morphine prevents the mitochon ...... nal pathway in cardiomyocytes.
@en
P2093
P2860
P304
P356
10.1152/AJPHEART.00453.2009
P577
2009-12-04T00:00:00Z