L-type Ca(2+) channel facilitation mediated by H(2)O(2)-induced activation of CaMKII in rat ventricular myocytes.
about
Ca(2+) signaling in the myocardium by (redox) regulation of PKA/CaMKIIRegulation of signal transduction by reactive oxygen species in the cardiovascular systemMitochondrial ion channels/transporters as sensors and regulators of cellular redox signaling.Revisiting the ionic mechanisms of early afterdepolarizations in cardiomyocytes: predominant by Ca waves or Ca currents?Mechanistic Investigation of the Arrhythmogenic Role of Oxidized CaMKII in the HeartRole of the transient outward potassium current in the genesis of early afterdepolarizations in cardiac cells.Docosahexaenoic Acid reduces the incidence of early afterdepolarizations caused by oxidative stress in rabbit ventricular myocytes.Hydrogen peroxide sensing and signaling by protein kinases in the cardiovascular system.Rotenone-stimulated superoxide release from mitochondrial complex I acutely augments L-type Ca2+ current in A7r5 aortic smooth muscle cells.Modeling CaMKII-mediated regulation of L-type Ca(2+) channels and ryanodine receptors in the heart.CaMKII in the cardiovascular system: sensing redox states.Modulating protein activity and cellular function by methionine residue oxidation.Redox control of cardiac excitability.Redox regulation of sodium and calcium handlingA Memory Molecule, Ca(2+)/Calmodulin-Dependent Protein Kinase II and Redox Stress; Key Factors for Arrhythmias in a Diseased Heart.Novel aspects of ROS signalling in heart failure.The promise of CaMKII inhibition for heart disease: preventing heart failure and arrhythmias.Mitochondrial Ca2+ and regulation of the permeability transition pore.Redox regulation of the actin cytoskeleton and its role in the vascular system.Nonspecific, reversible inhibition of voltage-gated calcium channels by CaMKII inhibitor CK59.Angiotensin II induces afterdepolarizations via reactive oxygen species and calmodulin kinase II signaling.Reactive oxygen species-activated Ca/calmodulin kinase IIδ is required for late I(Na) augmentation leading to cellular Na and Ca overload.Raised activity of L-type calcium channels renders neurons prone to form paroxysmal depolarization shifts.Ca/Calmodulin-dependent Protein Kinase II in Heart Failure.Sphingosylphosphorylcholine potentiates vasoreactivity and voltage-gated Ca2+ entry via NOX1 and reactive oxygen species.Mechanisms underlying the modulation of L-type Ca2+ channel by hydrogen peroxide in guinea pig ventricular myocytes.N-acetylcysteine prevents electrical remodeling and attenuates cellular hypertrophy in epicardial myocytes of rats with ascending aortic stenosis.Arrhythmogenic Mechanisms in Heart Failure: Linking β-Adrenergic Stimulation, Stretch, and CalciumMitochondrial-Mediated Oxidative Ca/Calmodulin-Dependent Kinase II Activation Induces Early Afterdepolarizations in Guinea Pig Cardiomyocytes: An In Silico Study
P2860
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P2860
L-type Ca(2+) channel facilitation mediated by H(2)O(2)-induced activation of CaMKII in rat ventricular myocytes.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
L-type Ca(2+) channel facilita ...... I in rat ventricular myocytes.
@en
type
label
L-type Ca(2+) channel facilita ...... I in rat ventricular myocytes.
@en
prefLabel
L-type Ca(2+) channel facilita ...... I in rat ventricular myocytes.
@en
P2093
P1476
L-type Ca(2+) channel facilita ...... II in rat ventricular myocytes
@en
P2093
Chung-Il Noh
Jin-Young Yoon
Shin-Young Ryu
Sun-Hyun Park
Won-Kyung Ho
Young-Hwan Song
P304
P356
10.1016/J.YJMCC.2009.10.020
P50
P577
2009-10-31T00:00:00Z