Diminished ICAM-1 expression and impaired pulmonary clearance of nontypeable Haemophilus influenzae in a mouse model of chronic obstructive pulmonary disease/emphysema.
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Lung microbiology and exacerbations in COPDModels of Respiratory Infections: Virus-Induced Asthma Exacerbations and BeyondIntratracheally administered titanium dioxide or carbon black nanoparticles do not aggravate elastase-induced pulmonary emphysema in ratsFrom in vitro to in vivo Models of Bacterial Biofilm-Related Infections.A short-term mouse model that reproduces the immunopathological features of rhinovirus-induced exacerbation of COPDNon-typeable Haemophilus influenzae invasion and persistence in the human respiratory tract.Dps promotes survival of nontypeable Haemophilus influenzae in biofilm communities in vitro and resistance to clearance in vivoRelative contributions of lipooligosaccharide inner and outer core modifications to nontypeable Haemophilus influenzae pathogenesis.Haemophilus parainfluenzae Strain ATCC 33392 Forms Biofilms In Vitro and during Experimental Otitis Media Infections.Respiratory syncytial virus persistence in macrophages downregulates intercellular adhesion molecule-1 expression and reduces adhesion of non-typeable Haemophilus influenzae.Insights on persistent airway infection by non-typeable Haemophilus influenzae in chronic obstructive pulmonary disease.Non-Typeable Haemophilus influenzae Infection of the Junbo Mouse.Emphysema induced by elastase enhances acute inflammatory pulmonary response to intraperitoneal LPS in rats.Peroxiredoxin-glutaredoxin and catalase promote resistance of nontypeable Haemophilus influenzae 86-028NP to oxidants and survival within neutrophil extracellular traps.LuxS promotes biofilm maturation and persistence of nontypeable haemophilus influenzae in vivo via modulation of lipooligosaccharides on the bacterial surface.Nontypeable Haemophilus influenzae initiates formation of neutrophil extracellular traps.Acute Lung Injury in Response to Intratracheal Instillation of Lipopolysaccharide in an Animal Model of Emphysema Induced by Elastase.Modulation of Haemophilus influenzae interaction with hydrophobic molecules by the VacJ/MlaA lipoprotein impacts strongly on its interplay with the airways.Revisiting cefditoren for the treatment of community-acquired infections caused by human-adapted respiratory pathogens in adults
P2860
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P2860
Diminished ICAM-1 expression and impaired pulmonary clearance of nontypeable Haemophilus influenzae in a mouse model of chronic obstructive pulmonary disease/emphysema.
description
2008 nî lūn-bûn
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2008年の論文
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2008年学术文章
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2008年学术文章
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name
Diminished ICAM-1 expression a ...... e pulmonary disease/emphysema.
@en
Diminished ICAM-1 expression a ...... e pulmonary disease/emphysema.
@nl
type
label
Diminished ICAM-1 expression a ...... e pulmonary disease/emphysema.
@en
Diminished ICAM-1 expression a ...... e pulmonary disease/emphysema.
@nl
prefLabel
Diminished ICAM-1 expression a ...... e pulmonary disease/emphysema.
@en
Diminished ICAM-1 expression a ...... e pulmonary disease/emphysema.
@nl
P2093
P2860
P356
P1476
Diminished ICAM-1 expression a ...... e pulmonary disease/emphysema.
@en
P2093
Nancy D Kock
Shayla L West-Barnette
W Edward Swords
Wenzhou Hong
P2860
P304
P356
10.1128/IAI.00664-08
P407
P577
2008-09-15T00:00:00Z