Lysophosphatidylcholine potentiates vascular contractile responses in rat aorta via activation of tyrosine kinase.

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Mechanisms underlying lysophosphatidylcholine-induced potentiation of vascular contractions in the Otsuka Long-Evans Tokushima Fatty (OLETF) rat aorta

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Q36054473-68AE6DF5-E580-4EE7-B27A-E994EAEE3294

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Lysophosphatidylcholine potentiates vascular contractile responses in rat aorta via activation of tyrosine kinase.

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http://www.wikidata.org/entity/Q43263936

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2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

@zh-cn

2002年学术文章

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2002年学术文章

@zh-my

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2002年學術文章

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2002年學術文章

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name

Lysophosphatidylcholine potent ...... activation of tyrosine kinase.

@en

Lysophosphatidylcholine potent ...... activation of tyrosine kinase.

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type

Item

label

Lysophosphatidylcholine potent ...... activation of tyrosine kinase.

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Lysophosphatidylcholine potent ...... activation of tyrosine kinase.

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Lysophosphatidylcholine potent ...... activation of tyrosine kinase.

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Lysophosphatidylcholine potent ...... activation of tyrosine kinase.

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Lysophosphatidylcholine potent ...... activation of tyrosine kinase.

@en

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Hiroshi Suenaga

http://www.w3.org/2001/XMLSchema#string

Katsuo Kamata

http://www.w3.org/2001/XMLSchema#string

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Signal transduction by receptors with tyrosine kinase activity

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Src tyrosine kinases and extracellular signal-regulated kinase 1/2 mitogen-activated protein kinases mediate pressure-induced c-fos expression in cannulated rat mesenteric small arteries.

Signal transduction mechanisms mediating the physiological and pathophysiological actions of angiotensin II in vascular smooth muscle cells.

Minimally modified low density lipoprotein stimulates monocyte endothelial interactions.

Oxidized low density lipoproteins cause contraction and inhibit endothelium-dependent relaxation in the pig coronary artery.

Epidermal growth factor, a vascular smooth muscle mitogen, induces rat aortic contraction.

Oxidatively modified low density lipoproteins: a potential role in recruitment and retention of monocyte/macrophages during atherogenesis.

Atherosclerosis or lipoprotein-induced endothelial dysfunction. Potential mechanisms underlying reduction in EDRF/nitric oxide activity.

Modification of low density lipoprotein by endothelial cells involves lipid peroxidation and degradation of low density lipoprotein phospholipids.

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789-799

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scholarly article

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10.1038/SJ.BJP.0704525

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English

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135

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2002-02-01T00:00:00Z

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11525467

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11834627

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aorta

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1573188

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Lysophosphatidylcholine potentiates vascular contractile responses in rat aorta via activation of tyrosine kinase.

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Lysophosphatidylcholine potentiates vascular contractile responses in rat aorta via activation of tyrosine kinase.

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