Mediators released from LPS-challenged lungs induce inflammatory responses in liver vascular endothelial cells and neutrophilic leukocytes.
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Mechanical ventilation with high tidal volumes attenuates myocardial dysfunction by decreasing cardiac edema in a rat model of LPS-induced peritonitisMechanisms of xenogeneic baboon platelet aggregation and phagocytosis by porcine liver sinusoidal endothelial cells.Effects of ventilation strategy on distribution of lung inflammatory cell activity.The role of intestinal mucosa injury induced by intra-abdominal hypertension in the development of abdominal compartment syndrome and multiple organ dysfunction syndrome.Patterns of blood protein concentrations of ELGANs classified by three patterns of respiratory disease in the first 2 postnatal weeksMonocyte Tumor Necrosis Factor-α-Converting Enzyme Catalytic Activity and Substrate Shedding in Sepsis and Noninfectious Systemic Inflammation.NOX2 in lung inflammation: quantum dot based in situ imaging of NOX2-mediated expression of vascular cell adhesion molecule-1The NLRP3 inflammasome is required for the development of hypoxemia in LPS/mechanical ventilation acute lung injury.Is there a role of TNFR1 in acute lung injury cases associated with extracorporeal circulation?Update on implications and mechanisms of angiogenesis in liver fibrosis.Renal cell carcinoma alters endothelial receptor expression responsible for leukocyte adhesion.Lung-derived mediators induce cytokine production in downstream organs via an NF-κB-dependent mechanism.Lung ventilation strategies and regional lung inflammation.18F-FDG kinetics parameters depend on the mechanism of injury in early experimental acute respiratory distress syndrome.The effect of tidal volume on systemic inflammation in Acid-induced lung injury.Iptakalim ameliorates monocrotaline-induced pulmonary arterial hypertension in rats.Emodin improves lipopolysaccharide-induced microcirculatory disturbance in rat mesentery.Diabetic ketoacidosis elicits systemic inflammation associated with cerebrovascular endothelial cell dysfunction.Lack of matrix metalloproteinase 3 in mouse models of lung injury ameliorates the pulmonary inflammatory response in female but not in male mice.
P2860
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P2860
Mediators released from LPS-challenged lungs induce inflammatory responses in liver vascular endothelial cells and neutrophilic leukocytes.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Mediators released from LPS-ch ...... s and neutrophilic leukocytes.
@en
Mediators released from LPS-ch ...... s and neutrophilic leukocytes.
@nl
type
label
Mediators released from LPS-ch ...... s and neutrophilic leukocytes.
@en
Mediators released from LPS-ch ...... s and neutrophilic leukocytes.
@nl
prefLabel
Mediators released from LPS-ch ...... s and neutrophilic leukocytes.
@en
Mediators released from LPS-ch ...... s and neutrophilic leukocytes.
@nl
P2093
P2860
P356
P1476
Mediators released from LPS-ch ...... ls and neutrophilic leukocytes
@en
P2093
L A McCaig
N Markovic
S Mizuguchi
P2860
P304
P356
10.1152/AJPGI.00278.2009
P577
2009-10-08T00:00:00Z