about
Kisspeptin directly stimulates gonadotropin-releasing hormone release via G protein-coupled receptor 54.Synthetic chemerin-derived peptides suppress inflammation through ChemR23Mll5 is required for normal spermatogenesisThe GPR54 gene as a regulator of pubertyThe orphan adhesion-GPCR GPR126 is required for embryonic development in the mouseInsulin-like peptide 5 is an orexigenic gastrointestinal hormoneNull mutation of the Lmo4 gene or a combined null mutation of the Lmo1/Lmo3 genes causes perinatal lethality, and Lmo4 controls neural tube development in miceSinus node dysfunction following targeted disruption of the murine cardiac sodium channel gene Scn5aSplenomegaly and modified erythropoiesis in KLF13-/- miceVariable Na(v)1.5 protein expression from the wild-type allele correlates with the penetrance of cardiac conduction disease in the Scn5a(+/-) mouse modelMechanistic insights into the more potent effect of KP-54 compared to KP-10 in vivo.Leptin-dependent neuronal NO signaling in the preoptic hypothalamus facilitates reproduction.The testosterone-dependent and independent transcriptional networks in the hypothalamus of Gpr54 and Kiss1 knockout male mice are not fully equivalentSalmonella typhi uses CFTR to enter intestinal epithelial cells.Inotropic action of the puberty hormone kisspeptin in rat, mouse and human: cardiovascular distribution and characteristics of the kisspeptin receptorInternational Union of Basic and Clinical Pharmacology. LXXVII. Kisspeptin receptor nomenclature, distribution, and function.Stella is a maternal effect gene required for normal early development in mice.Kiss1 mutant placentas show normal structure and function in the mouse.The link between nutritional status and insulin sensitivity is dependent on the adipocyte-specific peroxisome proliferator-activated receptor-gamma2 isoformDefending sperm functionPropranolol enhances cell cycle-related gene expression in pressure overloaded hearts.Arrhythmogenic mechanisms in the isolated perfused hypokalaemic murine heart.Hypogonadotropic hypogonadism in mice lacking a functional Kiss1 gene.Kisspeptin can stimulate gonadotropin-releasing hormone (GnRH) release by a direct action at GnRH nerve terminals.GPR54 and kisspeptins.Mice lacking pro-opiomelanocortin are sensitive to high-fat feeding but respond normally to the acute anorectic effects of peptide-YY(3-36).Does Kisspeptin Signaling have a Role in the Testes?The cyclin A1-CDK2 complex regulates DNA double-strand break repair.Mouse models to study the central regulation of puberty.The role of kisspeptin signaling in reproduction.Voltage dependence of the Ca(2+)-activated K(+) channel K(Ca)3.1 in human erythroleukemia cells.Comparison of protein transduction domains in mediating cell delivery of a secreted CRE protein.Intra-amniotic delivery of CFTR-expressing adenovirus does not reverse cystic fibrosis phenotype in inbred CFTR-knockout mice.Cystic fibrosis gene therapy.Aminoglycoside suppression of a premature stop mutation in a Cftr-/- mouse carrying a human CFTR-G542X transgene.Steroid hormone enhancement of gene delivery to a human airway epithelial cell line in vitro and mouse airways in vivo.Increased persistence of lung gene expression using plasmids containing the ubiquitin C or elongation factor 1alpha promoter.Enhanced gene delivery to human airway epithelial cells using an integrin-targeting lipoplex.Inflammation in cystic fibrosis airways: relationship to increased bacterial adherence.Hyperalgesic activity of kisspeptin in mice.
P50
Q24557494-6F4D1408-79BE-4C21-AF81-1436995BDFD8Q24656236-B2594DBB-9C7B-4ECE-A548-49FC179A8B19Q27313888-65561209-E2B3-415D-B0A6-C5D35464C282Q28211950-7EF9779D-56A7-46F0-B223-B905F84C790BQ28299700-4DC31C91-91A9-43F4-8E3A-3FCCE8B3B99EQ28507260-F21053B3-ADB9-43AD-B0C5-9FF74505EA85Q28587181-E08F8366-8507-4017-9326-FDB5C7FE7FC0Q28587978-E5C7480D-9D2D-4958-9845-3EBC8BC247E6Q28593191-B827A48E-46DB-436E-954E-CC9E38801B23Q33533311-65590736-985E-45C9-87AC-57B7560AAE81Q33626546-3201E77A-2C6C-4E22-89E2-246955EAEB61Q33872535-AA2DDD49-CB90-4DDB-AE48-A4900415E854Q33884157-ECB7A8E0-584C-4DBC-ABC0-BF358A70B70CQ34066552-B13C04FE-C3A6-4BCF-B014-6B264C5DE189Q34087759-72383C17-3D33-439F-97A9-61346ABCBAA9Q34149940-2708A731-A3DB-4DF0-929A-C1B47C68E778Q34280753-F2815A1D-AB36-4F9B-AD4D-609334C1C558Q34996680-6AB44F85-3C2E-4F2D-8BE2-B483BB0F8921Q35004514-C188554D-D7F2-4277-B4E8-B1EAE395B90CQ35034336-A386D69F-3271-4A31-A194-1747A24BDD62Q35635944-64E0B9F4-B922-41B0-90BE-DE202F3BC4A4Q35768779-086D7270-3522-4374-9C64-107156F04139Q35973482-F61A3A0F-1BD2-480F-AB79-93A2452A21F0Q36796644-F392C808-FDDD-4452-9A89-EF86E417B4B5Q37056265-33DF8646-6E06-4B67-A1E9-C025B4067BE1Q37358544-81E8AB3E-982A-4960-8F2F-A167852BF521Q37417911-1C77A3A5-15BE-451E-813D-B931C6F1B4A6Q37527220-4EF847EF-03B2-4C10-B9DB-6AAEB90C32FAQ37677253-91CDEB39-3C41-4B21-BF83-C6FF1927B81AQ37779175-841D30C2-0942-45CD-9714-DF7B2CA53F5DQ39194934-D16460C3-A33A-4D3E-8E02-5800DE6B2A1DQ40027828-EEE09FD4-9E50-4437-97E8-E647A4E87F60Q40106489-0F6E919A-1818-46EF-8F8A-493743094896Q40392891-B5232B70-E135-4C30-ABF6-E16BAC44BF9EQ40704575-0C79D8FE-9320-4189-955B-3318EDEF7643Q40769262-7866CBAD-08B8-45E7-987F-BDC2444FE66CQ40769266-2D971A9D-D58E-4996-8087-AB43EE8F7344Q40810576-37861778-0C8F-4A33-A6D5-9039797C13BEQ40812163-A13A6ECF-D379-463E-9F21-EE15F2210122Q41439649-F15A4B75-A917-4E2D-9C16-EF13F15E5D90
P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
William H. Colledge
@ast
William H. Colledge
@en
William H. Colledge
@es
William H. Colledge
@nl
William H. Colledge
@sl
type
label
William H. Colledge
@ast
William H. Colledge
@en
William H. Colledge
@es
William H. Colledge
@nl
William H. Colledge
@sl
prefLabel
William H. Colledge
@ast
William H. Colledge
@en
William H. Colledge
@es
William H. Colledge
@nl
William H. Colledge
@sl
P106
P1153
7006668544
P21
P2798
P31
P496
0000-0002-9603-4429