Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
about
The mucosal immune system in the oral cavity-an orchestra of T cell diversityRegulatory T Cells Control Th2-Dominant Murine Autoimmune GastritisIntestinal microbiota link lymphopenia to murine autoimmunity via PD-1+CXCR5-/dim B-helper T cell inductionThe Th17 pathway as a therapeutic target in rheumatoid arthritis and other autoimmune and inflammatory disorders.New insights into the role of T cells in pathogenesis of psoriasis and psoriatic arthritis.Feedback inhibition of CREB signaling promotes beta cell dysfunction in insulin resistance.Association Between IL-17, IL-8 and IL-18 Expression in Peripheral Blood and Helicobacter Pylori Infection in Mongolian Gerbils.Interleukin-21 and tumor necrosis factor-α are critical for the development of autoimmune gastritis in mice.
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P2860
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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name
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
@en
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
@nl
type
label
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
@en
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
@nl
prefLabel
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
@en
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
@nl
P2093
P2860
P50
P356
P1476
Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.
@en
P2093
Desmond K Y Ang
Michele W L Teng
Shayne A Bellingham
P2860
P304
P356
10.1002/EJI.201142341
P407
P577
2012-08-15T00:00:00Z