Tamoxifen inhibits nitrotyrosine formation after reversible middle cerebral artery occlusion in the rat.
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Nitric oxide and peroxynitrite in health and diseaseAneurysmal Subarachnoid Hemorrhage and Neuroinflammation: A Comprehensive ReviewEvidence-based pharmacotherapy for cerebral vasospasm.Astrocyte-derived growth factors and estrogen neuroprotection: role of transforming growth factor-α in estrogen-induced upregulation of glutamate transporters in astrocytesTamoxifen and estradiol improved locomotor function and increased spared tissue in rats after spinal cord injury: their antioxidant effect and role of estrogen receptor alpha.Volume-regulated anion channel--a frenemy within the brain.Protective effects of estrogen and selective estrogen receptor modulators in the brain.Neuroprotection by tamoxifen in focal cerebral ischemia is not mediated by an agonist action at estrogen receptors but is associated with antioxidant activity.Neurotrophic and neuroprotective actions of estrogen: basic mechanisms and clinical implications.Tamoxifen neuroprotection in cerebral ischemia involves attenuation of kinase activation and superoxide production and potentiation of mitochondrial superoxide dismutase.Disruption of ionic and cell volume homeostasis in cerebral ischemia: The perfect stormAstrocyte dysfunction in neurological disorders: a molecular perspective.Tamoxifen mediated estrogen receptor activation protects against early impairment of hippocampal neuron excitability in an oxygen/glucose deprivation brain slice ischemia model.cAMP response element-binding protein (CREB) and nuclear factor κB mediate the tamoxifen-induced up-regulation of glutamate transporter 1 (GLT-1) in rat astrocytesTamoxifen as an effective neuroprotectant against early brain injury and learning deficits induced by subarachnoid hemorrhage: possible involvement of inflammatory signaling.Selective vulnerability of synaptic signaling and metabolism to nitrosative stressTamoxifen reduces infiltration of inflammatory cells, apoptosis and inhibits IKK/NF-kB pathway after spinal cord injury in rats.Tamoxifen Administration Immediately or 24 Hours after Spinal Cord Injury Improves Locomotor Recovery and Reduces Secondary Damage in Female Rats.DCPIB, a specific inhibitor of volume regulated anion channels (VRACs), reduces infarct size in MCAo and the release of glutamate in the ischemic cortical penumbra.Peroxynitrite enhances astrocytic volume-sensitive excitatory amino acid release via a src tyrosine kinase-dependent mechanism.Hypoosmotic swelling increases protein tyrosine nitration in cultured rat astrocytes.The levels of glutathione and nitrite-nitrate and the expression of Bcl-2 mRNA in ovariectomized rats treated by raloxifene against kainic acid.DJ-1 protects against neurodegeneration caused by focal cerebral ischemia and reperfusion in rats.
P2860
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P2860
Tamoxifen inhibits nitrotyrosine formation after reversible middle cerebral artery occlusion in the rat.
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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name
Tamoxifen inhibits nitrotyrosi ...... l artery occlusion in the rat.
@en
Tamoxifen inhibits nitrotyrosi ...... l artery occlusion in the rat.
@nl
type
label
Tamoxifen inhibits nitrotyrosi ...... l artery occlusion in the rat.
@en
Tamoxifen inhibits nitrotyrosi ...... l artery occlusion in the rat.
@nl
prefLabel
Tamoxifen inhibits nitrotyrosi ...... l artery occlusion in the rat.
@en
Tamoxifen inhibits nitrotyrosi ...... l artery occlusion in the rat.
@nl
P2093
P2860
P1476
Tamoxifen inhibits nitrotyrosi ...... l artery occlusion in the rat.
@en
P2093
B I Tranmer
H K Kimelberg
P J Feustel
P2860
P304
P356
10.1046/J.1471-4159.2001.00198.X
P407
P577
2001-03-01T00:00:00Z