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Protective effect of vasoactive intestinal peptide on bone destruction in the collagen-induced arthritis model of rheumatoid arthritis.Neuroendocrine differentiation of the LNCaP prostate cancer cell line maintains the expression and function of VIP and PACAP receptorsDifferential expression of vasoactive intestinal peptide and its functional receptors in human osteoarthritic and rheumatoid synovial fibroblastsThe pathogenic Th profile of human activated memory Th cells in early rheumatoid arthritis can be modulated by VIP.Regulation of TLR expression, a new perspective for the role of VIP in immunity.VIP and CRF reduce ADAMTS expression and function in osteoarthritis synovial fibroblasts.Peptides targeting Toll-like receptor signalling pathways for novel immune therapeutics.Vasoactive intestinal peptide increases vascular endothelial growth factor expression and neuroendocrine differentiation in human prostate cancer LNCaP cells.VIP reverses the expression profiling of TLR4-stimulated signaling pathway in rheumatoid arthritis synovial fibroblasts.VIP down-regulates TLR4 expression and TLR4-mediated chemokine production in human rheumatoid synovial fibroblasts.Effect of VIP on the balance between cytokines and master regulators of activated helper T cells.Coexpression of AT1 and AT2 receptors by human fibroblasts is associated with resistance to angiotensin II.Inflammatory mediators alter interleukin-17 receptor, interleukin-12 and -23 expression in human osteoarthritic and rheumatoid arthritis synovial fibroblasts: immunomodulation by vasoactive intestinal Peptide.Healthy and Osteoarthritic Synovial Fibroblasts Produce a Disintegrin and Metalloproteinase with Thrombospondin Motifs 4, 5, 7, and 12: Induction by IL-1β and Fibronectin and Contribution to Cartilage Damage.VIP impairs acquisition of the macrophage proinflammatory polarization profile.IL-22/IL-22R1 axis and S100A8/A9 alarmins in human osteoarthritic and rheumatoid arthritis synovial fibroblasts.Mapping the CRF-urocortins system in human osteoarthritic and rheumatoid synovial fibroblasts: effect of vasoactive intestinal peptide.RNA sensors in human osteoarthritis and rheumatoid arthritis synovial fibroblasts: immune regulation by vasoactive intestinal peptide.VIP modulates IL-22R1 expression and prevents the contribution of rheumatoid synovial fibroblasts to IL-22-mediated joint destruction.VIP decreases TLR4 expression induced by LPS and TNF-alpha treatment in human synovial fibroblastsTopical application of a peptide inhibitor of transforming growth factor-beta1 ameliorates bleomycin-induced skin fibrosisImmunoregulatory properties of vasoactive intestinal peptide in human T cell subsets: implications for rheumatoid arthritisNew insights into the role of VIP on the ratio of T-cell subsets during the development of autoimmune diabetesCXCL12 gene expression is upregulated by hypoxia and growth arrest but not by inflammatory cytokines in rheumatoid synovial fibroblastsVasoactive intestinal peptide maintains the nonpathogenic profile of human th17-polarized cellsA Clinical Approach for the Use of VIP Axis in Inflammatory and Autoimmune DiseasesProfile of Matrix-Remodeling Proteinases in Osteoarthritis: Impact of Fibronectin
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Irene Gutiérrez-Cañas
@ast
Irene Gutiérrez-Cañas
@en
Irene Gutiérrez-Cañas
@es
Irene Gutiérrez-Cañas
@nl
Irene Gutiérrez-Cañas
@sl
type
label
Irene Gutiérrez-Cañas
@ast
Irene Gutiérrez-Cañas
@en
Irene Gutiérrez-Cañas
@es
Irene Gutiérrez-Cañas
@nl
Irene Gutiérrez-Cañas
@sl
prefLabel
Irene Gutiérrez-Cañas
@ast
Irene Gutiérrez-Cañas
@en
Irene Gutiérrez-Cañas
@es
Irene Gutiérrez-Cañas
@nl
Irene Gutiérrez-Cañas
@sl
P108
P106
P21
P31
P496
0000-0003-0678-3619