An examination of the effects of osmotic pressure changes upon transmitter release from mammalian motor nerve terminals.
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Cell biology of Ca2+-triggered exocytosisHypertonicity-induced transmitter release at Drosophila neuromuscular junctions is partly mediated by integrins and cAMP/protein kinase AHypertonic enhancement of transmitter release from frog motor nerve terminals: Ca2+ independence and role of integrinsHyperosmolarity reduces facilitation by a Ca(2+)-independent mechanism at the lobster neuromuscular junction: possible depletion of the releasable poolReduced expression of the vesicular acetylcholine transporter and neurotransmitter content affects synaptic vesicle distribution and shape in mouse neuromuscular junction.External Ca(2+)-independent release of neurotransmitters.Readily releasable pool size changes associated with long term depressionChanges in the structure of neuromuscular junctions caused by variations in osmotic pressure.On the inhibition of muscle contraction caused by exposure to hypertonic solutionsRemoval of extracellular chloride suppresses transmitter release from photoreceptor terminals in the mudpuppy retina.Evidence for a voltage-dependent enhancement of neurotransmitter release mediated via the synaptic protein interaction site of N-type Ca2+ channels.Inosine induces presynaptic inhibition of acetylcholine release by activation of A3 adenosine receptors at the mouse neuromuscular junction.Spontaneous release of transmitter from 'repressed' nerve terminals in axolotl muscle.A cyclic adenosine monophosphate link in the catecholamine enhancement of transmitter release at the neuromuscular junction.Insulin increases amino acid transport into rat soleus motor axons.The contractile response of smooth muscle to immersion in hypertonic solutions.Adenosine depresses a Ca(2+)-independent step in transmitter exocytosis at frog motor nerve terminals.Biphasic modulation of synaptic transmission by hypertonicity at the embryonic Drosophila neuromuscular junction.Cardiovascular changes induced by central hypertonic saline are accompanied by glutamate release in awake rats.Thyroid hormones regulate the frequency of miniature end-plate currents in pre- and prometamorphic stages of the tadpole tail.The possible role of fixed membrane surface charges in acetylcholine release at the frog neuromuscular junction.Influence of lanthanum on transmitter release at the neuromuscular junction.Supraoptic neurones of rat hypothalamus are osmosensitiveBiological Sciences: Release of ATP from Rat Motor Nerve Terminals
P2860
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P2860
An examination of the effects of osmotic pressure changes upon transmitter release from mammalian motor nerve terminals.
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1968 nî lūn-bûn
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1968年の論文
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1968年学术文章
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1968年学术文章
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1968年学术文章
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1968年学术文章
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1968年学术文章
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1968年學術文章
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1968年學術文章
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name
An examination of the effects ...... mmalian motor nerve terminals.
@en
An examination of the effects ...... mmalian motor nerve terminals.
@nl
type
label
An examination of the effects ...... mmalian motor nerve terminals.
@en
An examination of the effects ...... mmalian motor nerve terminals.
@nl
prefLabel
An examination of the effects ...... mmalian motor nerve terminals.
@en
An examination of the effects ...... mmalian motor nerve terminals.
@nl
P2093
P1476
An examination of the effects ...... mmalian motor nerve terminals.
@en
P2093
P304
P356
10.1113/JPHYSIOL.1968.SP008579
P407
P577
1968-08-01T00:00:00Z