The maxi-K channel opener BMS-204352 attenuates regional cerebral edema and neurologic motor impairment after experimental brain injury.
about
Voltage-gated cation channel modulators for the treatment of strokeThe science of cerebral ischemia and the quest for neuroprotection: navigating past failure to future successBench to bedside: brain edema and cerebral resuscitation: the present and future.Down-regulation of BK channel expression in the pilocarpine model of temporal lobe epilepsy.Delayed neuronal preconditioning by NS1619 is independent of calcium activated potassium channels.Critical role of large-conductance calcium- and voltage-activated potassium channels in leptin-induced neuroprotection of N-methyl-d-aspartate-exposed cortical neurons.Mitochondrial mechanisms in cerebral vascular control: shared signaling pathways with preconditioning.BK channel activators and their therapeutic perspectivesComputational models of neuronal biophysics and the characterization of potential neuropharmacological targets.Cognitive evaluation of traumatically brain-injured rats using serial testing in the Morris water maze.The novel antiepileptic agent RWJ-333369-A, but not its analog RWJ-333369, reduces regional cerebral edema without affecting neurobehavioral outcome or cell death following experimental traumatic brain injuryProtective effects of cilostazol against transient focal cerebral ischemia and chronic cerebral hypoperfusion injury.Mitochondrial-mediated suppression of ROS production upon exposure of neurons to lethal stress: mitochondrial targeted preconditioning.Modulation of BKCa channel gating by endogenous signaling molecules.CO metabolism, sensing, and signaling.The need for new approaches in CNS drug discovery: Why drugs have failed, and what can be done to improve outcomes.Molecular Determinants of BK Channel Functional Diversity and Functioning.In vitro protein binding studies with BMS-204352: lack of protein binding displacement interaction in human serum.Local potassium signaling couples neuronal activity to vasodilation in the brain.Effect of dose and input rate on the brain penetration of BMS-204352 following intravenous administration to rats.Role of Mitochondria in Cerebral Vascular Function: Energy Production, Cellular Protection, and Regulation of Vascular Tone.The effects of exposure to 915 MHz radiofrequency identification on cerebral glucose metabolism in rat: a [F-18] FDG micro-PET study.
P2860
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P2860
The maxi-K channel opener BMS-204352 attenuates regional cerebral edema and neurologic motor impairment after experimental brain injury.
description
2001 nî lūn-bûn
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2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
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2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
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2001年學術文章
@yue
2001年學術文章
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name
The maxi-K channel opener BMS- ...... ter experimental brain injury.
@en
The maxi-K channel opener BMS- ...... ter experimental brain injury.
@nl
type
label
The maxi-K channel opener BMS- ...... ter experimental brain injury.
@en
The maxi-K channel opener BMS- ...... ter experimental brain injury.
@nl
prefLabel
The maxi-K channel opener BMS- ...... ter experimental brain injury.
@en
The maxi-K channel opener BMS- ...... ter experimental brain injury.
@nl
P2093
P2860
P1476
The maxi-K channel opener BMS- ...... ter experimental brain injury.
@en
P2093
Bareyre FM
Gribkoff V
McIntosh TK
Raghupathi R
Saatman KE
Starrett JE Jr
Weisser JD
P2860
P304
P356
10.1097/00004647-200104000-00008
P577
2001-04-01T00:00:00Z
P5875
P6179
1053050135