Both caspase-dependent and caspase-independent pathways may be involved in hippocampal CA1 neuronal death because of loss of cytochrome c From mitochondria in a rat forebrain ischemia model.
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Bcl-2 transfection via herpes simplex virus blocks apoptosis-inducing factor translocation after focal ischemia in the ratBoth ischemic preconditioning and ghrelin administration protect hippocampus from ischemia/reperfusion and upregulate uncoupling protein-2Effects of CXCR4 gene transfer on cardiac function after ischemia-reperfusion injury.Effects of transplanted GDNF gene modified marrow stromal cells on focal cerebral ischemia in rats.Ischaemic preconditioning: therapeutic implications for stroke?Mechanisms of neural cell death: implications for development of neuroprotective treatment strategiesGene activation and protein expression following ischaemic stroke: strategies towards neuroprotection.Apoptosis-inducing factor is involved in the regulation of caspase-independent neuronal cell death.Mechanisms of early brain injury after subarachnoid hemorrhage.Cell death atlas of the postnatal mouse ventral forebrain and hypothalamus: effects of age and sex.Ischemic preconditioning targets the respiration of synaptic mitochondria via protein kinase C epsilon.Temporal regulation of apoptotic and anti-apoptotic molecules after middle cerebral artery occlusion followed by reperfusionFAK/Src family of kinases: protective or aggravating factor for ischemia reperfusion injury in nervous system?Altered apoptotic responses in neurons lacking RhoB GTPase.Patterns of cell death in the perinatal mouse forebrain.Ischemic postconditioning may not influence early brain injury induced by focal cerebral ischemia/reperfusion in rats.Activation of p38 mitogen-activated protein kinase and partial reactivation of the cell cycle by cis-4-methylsphingosine direct postmitotic neurons towards apoptosis.Cytochrome c associated apoptosis during ATP recovery after hypoxia in neonatal rat cerebrocortical slices.Ischemia leads to apoptosis--and necrosis-like neuron death in the ischemic rat hippocampus.Depressed responses to applied and synaptically-released GABA in CA1 pyramidal cells, but not in CA1 interneurons, after transient forebrain ischemia.Inhibition of caspase-9 activation and apoptosis is involved in ischemic preconditioning-induced neuroprotection in rat brain.Impaired firing and sodium channel function in CA1 hippocampal interneurons after transient cerebral ischemia.Prevention of apoptosis-inducing factor translocation is a possible mechanism for protective effects of hepatocyte growth factor against neuronal cell death in the hippocampus after transient forebrain ischemia.
P2860
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P2860
Both caspase-dependent and caspase-independent pathways may be involved in hippocampal CA1 neuronal death because of loss of cytochrome c From mitochondria in a rat forebrain ischemia model.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
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2001年學術文章
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name
Both caspase-dependent and cas ...... rat forebrain ischemia model.
@en
Both caspase-dependent and cas ...... rat forebrain ischemia model.
@nl
type
label
Both caspase-dependent and cas ...... rat forebrain ischemia model.
@en
Both caspase-dependent and cas ...... rat forebrain ischemia model.
@nl
prefLabel
Both caspase-dependent and cas ...... rat forebrain ischemia model.
@en
Both caspase-dependent and cas ...... rat forebrain ischemia model.
@nl
P2093
P2860
P1476
Both caspase-dependent and cas ...... rat forebrain ischemia model.
@en
P2093
Fujihara H
P2860
P304
P356
10.1097/00004647-200105000-00007
P577
2001-05-01T00:00:00Z
P5875
P6179
1017219776