Persistent E-cadherin expression in inflammatory breast cancer. - Wikidata - awgldk - TriplyDB

Persistent E-cadherin expression in inflammatory breast cancer.

Persistent E-cadherin expression in inflammatory breast cancer.

http://www.wikidata.org/entity/Q43607916

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Inflammatory breast cancer: clinical progress and the main problems that must be addressed Monocytes conditioned media stimulate fibronectin expression and spreading of inflammatory breast cancer cells in three-dimensional culture: A mechanism mediated by IL-8 signaling pathway Characterization of RhoC expression in benign and malignant breast disease: a potential new marker for small breast carcinomas with metastatic ability Update on inflammatory breast cancer In vitro analysis of the invasive phenotype of SUM 149, an inflammatory breast cancer cell line E-cadherin expression in primary carcinomas of the breast and its distant metastases E-cadherin's dark side: possible role in tumor progression Adhesion molecules in breast carcinoma: a challenge to the pathologist E-cadherin and gastric cancer: cause, consequence, and applications Inflammatory breast cancer: New factors contribute to disease etiology: A review Inflammatory breast cancer: what we know and what we need to learn A Novel Role of E-Cadherin-Based Adherens Junctions in Neoplastic Cell Dissemination Lgl2 executes its function as a tumor suppressor by regulating ErbB signaling in the zebrafish epidermis Adhesion in mammary development: novel roles for E-cadherin in individual and collective cell migration Targeting GLI1 expression in human inflammatory breast cancer cells enhances apoptosis and attenuates migration.A stromal gene signature associated with inflammatory breast cancer.Epigallocatechin-3-gallate inhibits stem-like inflammatory breast cancer cells Gain in cellular organization of inflammatory breast cancer: A 3D in vitro model that mimics the in vivo metastasis Mesenchymal stem cells promote mammosphere formation and decrease E-cadherin in normal and malignant breast cells.Challenging a Misnomer? The Role of Inflammatory Pathways in Inflammatory Breast Cancer Inflammatory breast cancer: a model for investigating cluster-based dissemination.High-resolution comparative genomic hybridization of inflammatory breast cancer and identification of candidate genes.EMT and MET: necessary or permissive for metastasis?Involvement of members of the cadherin superfamily in cancer Rab11 regulates E-cadherin expression and induces cell transformation in colorectal carcinoma.Expression of autotaxin and lysophosphatidic acid receptors increases mammary tumorigenesis, invasion, and metastases.An immunohistochemically positive E-cadherin status is not always predictive for a good prognosis in human breast cancer A core invasiveness gene signature reflects epithelial-to-mesenchymal transition but not metastatic potential in breast cancer cell lines and tissue samples.CDH22 expression is reduced in metastatic melanoma.Triple-negative subtype predicts poor overall survival and high locoregional relapse in inflammatory breast cancer Inflammation Mediated Metastasis: Immune Induced Epithelial-To-Mesenchymal Transition in Inflammatory Breast Cancer Cells Mechanobiology of tumor invasion: engineering meets oncology.Cadherin cell adhesion system in canine mammary cancer: a review.A Preclinical Model of Inflammatory Breast Cancer to Study the Involvement of CXCR4 and ACKR3 in the Metastatic Process.Expression of epithelial to mesenchymal transition-related markers in lymph node metastases as a surrogate for primary tumor metastatic potential in breast cancer.Real time PCR analyses of expression of E-cadherin, alpha-, beta- and gamma-catenin in human breast cancer for predicting clinical outcome.CCN6 (WISP3) as a new regulator of the epithelial phenotype in breast cancer.Secretion of N- and O-linked Glycoproteins from 4T1 Murine Mammary Carcinoma Cells Comparison of the effects of phenethyl isothiocyanate and sulforaphane on gene expression in breast cancer and normal mammary epithelial cells.CDH22 hypermethylation is an independent prognostic biomarker in breast cancer.

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Persistent E-cadherin expression in inflammatory breast cancer.

http://www.wikidata.org/entity/Q43607916

description

2001 nî lūn-bûn

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2001年の論文

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年學術文章

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2001年學術文章

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2001年學術文章

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name

Persistent E-cadherin expression in inflammatory breast cancer.

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Persistent E-cadherin expression in inflammatory breast cancer.

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type

label

Persistent E-cadherin expression in inflammatory breast cancer.

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Persistent E-cadherin expression in inflammatory breast cancer.

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prefLabel

Persistent E-cadherin expression in inflammatory breast cancer.

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Persistent E-cadherin expression in inflammatory breast cancer.

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Modern Pathology

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Persistent E-cadherin expression in inflammatory breast cancer.

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Braun T

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Kleer CG

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Merajver SD

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P2860

A causal role for E-cadherin in the transition from adenoma to carcinoma

Cadherin cell adhesion receptors as a morphogenetic regulator

The role of the cell-adhesion molecule E-cadherin as a tumour-suppressor gene.

Methylation patterns of the E-cadherin 5' CpG island are unstable and reflect the dynamic, heterogeneous loss of E-cadherin expression during metastatic progression.

Inflammatory breast cancer: a review.

Expression cloning of oncogenes by retroviral transfer of cDNA libraries.

Signal transduction through beta-catenin and specification of cell fate during embryogenesis.

Truncation of the beta-catenin binding domain of E-cadherin precedes epithelial apoptosis during prostate and mammary involution.

E-cadherin mediates aggregation-dependent survival of prostate and mammary epithelial cells through the retinoblastoma cell cycle control pathway.

Locally advanced breast cancer with inflammatory component: a clinical entity with a poor prognosis.

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